-
2
-
-
0028147382
-
ICP34.5 mutants of herpes simplex virus type 1 strain 17syn+ are attenuated for neurovirulence in mice and for replication in confluent primary mouse embryo cell culture
-
(1994)
J. Virol.
, vol.68
, pp. 48-55
-
-
Bolovan, C.A.1
Sawtell, N.M.2
Thompson, R.L.3
-
3
-
-
0024465766
-
Herpes simplex virus type 1 ICP0 plays a critical role in the de novo synthesis of infectious virus following transfection of viral DNA
-
(1989)
J. Virol.
, vol.63
, pp. 4570-4589
-
-
Cai, W.1
Schaffer, P.A.2
-
4
-
-
0031710037
-
The herpes simplex virus US11 protein effectively compensates for the gamma1(34.5) gene if present before activation of protein kinase R by precluding its phosphorylation and that of the alpha subunit of eukaryotic translation initiation factor 2
-
(1998)
J. Virol.
, vol.72
, pp. 8620-8626
-
-
Cassady, K.A.1
Gross, M.2
Roizman, B.3
-
5
-
-
0033611590
-
Herpes virus induced proteosome-dependent degradation of the nuclear bodies-associated PML and Sp100 proteins
-
(1999)
Oncogene
, vol.18
, pp. 935-941
-
-
Chelbi-Alix, M.K.1
DeThe, H.2
-
6
-
-
0028970730
-
Association of a M(r) 90,000 phosphoprotein with protein kinase PKR in cells exhibiting enhanced phosphorylation of translation initiation facor eIF-2 alpha and premature shutoff of protein synthesis after infection with gamma 1 34.5 mutants of herpes simplex virus 1
-
(1995)
Proc. Natl. Acad. Sci. USA
, vol.92
, pp. 10516-10520
-
-
Chou, J.1
Chen, J.J.2
Gross, M.3
Roizman, B.4
-
8
-
-
0026539149
-
The gamma 1(34.5) gene of herpes simplex virus1 precludes neuroblastoma cells from triggering total shutoff of protein synthesis characteristic of programmed cell death in neuronal cells
-
(1992)
Proc. Natl. Acad. Sci. USA
, vol.89
, pp. 3266-3270
-
-
Chou, J.1
Roizman, B.2
-
9
-
-
0028277376
-
Herpes simplex virus 1 gamma(1)34.5 gene function, which blocks the host response to infection, maps in the homologous domain of the genes expressed during growth arrest and DNA damage
-
(1994)
Proc. Natl. Acad. Sci. USA
, vol.91
, pp. 5247-5251
-
-
Chou, J.1
Roizman, B.2
-
11
-
-
0033624137
-
ICP0, a regulator of herpes simplex virus during lytic and latent infection
-
(2000)
BioEssays
, vol.22
, pp. 761-770
-
-
Everett, R.D.1
-
14
-
-
0345164384
-
Molecular mechanisms of interferon resistance mediated by viral-directed inhibition of PKR, the interferon-induced protein kinase
-
(1998)
Pharmacol. Ther.
, vol.78
, pp. 29-46
-
-
Gale, M.1
Katze, M.G.2
-
18
-
-
0031017382
-
134.5 protein of herpes simplex virus 1 complexes with protein phosphatase 1 a to dephosphorylate the α subunit of the eukaryotic translation inititation factor 2 and preclude the shutoff of protein synthesis by double-stranded RNA-activated protein kinase
-
(1997)
Proc. Natl. Acad. Sci. USA
, vol.94
, pp. 843-848
-
-
He, B.1
Gross, M.2
Roizman, B.3
-
25
-
-
0018876458
-
Inhibition of herpes simplex virus type 1 specific translation in cells treated with poly(rI) · poly(rC)
-
(1980)
J. Gen. Virol.
, vol.47
, pp. 97-111
-
-
Lipp, M.1
Brandner, G.2
-
26
-
-
0032143446
-
Nuclear domain 10, the site of DNA virus transcription and replication
-
(1998)
BioEssays
, vol.20
, pp. 660-667
-
-
Maul, G.G.1
-
31
-
-
0344299239
-
Viral immediate-early proteins abrogate the modification by SUMO-1 of PML and Sp100 proteins, correlating with nuclear body disruption
-
(1999)
J. Virol.
, vol.73
, pp. 5137-5143
-
-
Muller, S.1
Dejean, A.2
-
32
-
-
0029814850
-
Inhibition of herpes simplex virus type 1 immediate-early gene expression by alpha interferon is not VP16 specific
-
(1996)
J. Virol.
, vol.70
, pp. 6336-6339
-
-
Nicholl, M.J.1
Preston, C.M.2
-
35
-
-
0000595684
-
Herpes simplex viruses and their replication
-
B. N. Fields, D. M. Knipe, P. M. Howley, et al. (ed.), Lippincott-Raven, Philadelphia, Pa
-
(1996)
Fundamental virology, 3rd ed.
, pp. 1043-1107
-
-
Roizman, B.1
Sears, A.E.2
-
37
-
-
0029102140
-
An activity specified by the osteosarcoma line U2OS can substitute functionally for ICP0, a major regulatory protein of herpes simplex virus type 1
-
(1995)
J. Virol.
, vol.69
, pp. 6249-6258
-
-
Yao, F.1
Schaffer, P.A.2
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