Increased soluble thrombomodulin does not always indicate endothelial injury [1]

Clinical and Applied Thrombosis/Hemostasis

Volumn 8, Issue 1, 2002, Pages 87-89

  Borawski, Jacek ^a   Naumnik, Beata ^a   Myśliwiec, Michał ^a  

^a MEDICAL UNIVERSITY OF BIALYSTOK   (Poland)

Author keywords

[No Author keywords available]

Indexed keywords

ALANINE AMINOTRANSFERASE; THROMBOMODULIN;

ALANINE AMINOTRANSFERASE BLOOD LEVEL; HEMATOLOGICAL PARAMETERS; HEMODIALYSIS; HEMOLYTIC UREMIC SYNDROME; HUMAN; KIDNEY FAILURE; KIDNEY FUNCTION; LETTER; LIVER FUNCTION; PRIORITY JOURNAL; THROMBOTIC THROMBOCYTOPENIC PURPURA;

EID: 0036110515     PISSN: 10760296     EISSN: None     Source Type: Journal    
DOI: 10.1177/107602960200800113     Document Type: Letter

References (14)

1. Increased plasma thrombomodulin as a vascular endothelial cell marker in patients with thrombotic thrombocytopenic purpura and hemolytic uremic syndrome
2. Plasma thrombomodulin in health and diseases
3. Evaluation of plasma thrombomodulin (TM) levels in patients with liver disease
4. Thrombomodulin expression of sinusoidal endothelial cells in chronic viral hepatitis
5. Increased serum levels and sinusoidal expression of thrombomodulin in acute liver damage
6. Soluble thrombomodulin is associated with viral hepatitis, blood pressure, and medications in haemodialysis patients
7. Effluent levels of thrombomodulin predict early graft function in clinical liver transplantation
8. Thrombomodulin: A marker for endothelial damage during orthoptic liver transplantation
9. Soluble thrombomodulin antigen as a marker for endothelial damage during liver transplantation
10. A role for thrombomodulin in the pathogenesis of thrombin-induced thromboembolism in mice
11. Plasma thrombomodulin levels are dependent on renal function
12. Endothelial cell markers in chronic uremia: Relationship with hemostatic defects and severity of renal failure
13. Renal function, proteinuria and ACE-inhibitor therapy as determinants of plasma levels of endothelial markers
14. Hepatic response to sepsis: Interaction between coagulation and inflammatory processes