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Volumn 22, Issue 11, 2002, Pages 3663-3673
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The transcription factor B-Myb is maintained in an inhibited state in target cells through its interaction with the nuclear corepressors N-CoR and SMRT
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Author keywords
[No Author keywords available]
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Indexed keywords
CYCLIN A;
NUCLEAR RECEPTOR COREPRESSOR;
PROTEIN SMRT;
REPRESSOR PROTEIN;
TRANSCRIPTION FACTOR;
TRANSCRIPTION FACTOR B MYB;
UNCLASSIFIED DRUG;
ANIMAL CELL;
ARTICLE;
BINDING SITE;
CELL CYCLE;
CONTROLLED STUDY;
GENE EXPRESSION REGULATION;
GENE MUTATION;
MOUSE;
NONHUMAN;
PRIORITY JOURNAL;
PROTEIN PROTEIN INTERACTION;
TRANSCRIPTION REGULATION;
ANIMALS;
CDC2-CDC28 KINASES;
CELL CYCLE PROTEINS;
CELL LINE;
CYCLIN A;
CYCLIN-DEPENDENT KINASE 2;
CYCLIN-DEPENDENT KINASES;
DNA-BINDING PROTEINS;
MEMBRANE PROTEINS;
MICE;
MODELS, BIOLOGICAL;
MUTATION;
NUCLEAR PROTEINS;
PHOSPHOPROTEINS;
PHOSPHORYLATION;
PROTEIN STRUCTURE, TERTIARY;
PROTEIN-SERINE-THREONINE KINASES;
PROTO-ONCOGENE PROTEINS C-MYB;
RECOMBINANT FUSION PROTEINS;
REPRESSOR PROTEINS;
TRANS-ACTIVATORS;
TRANSCRIPTION, GENETIC;
ANIMALIA;
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EID: 0036097193
PISSN: 02707306
EISSN: None
Source Type: Journal
DOI: 10.1128/MCB.22.11.3663-3673.2002 Document Type: Article |
Times cited : (33)
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References (37)
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