Movement of toxic granulation neutrophils and C-reactive protein in inflammatory processes [7]

American Journal of Medicine

Volumn 112, Issue 7, 2002, Pages 595-596

  Kabutomori, Osamu ^a   Kanakura, Yuzuru ^a   Iwatani, Yoshinori ^b  

^a OSAKA UNIVERSITY HOSPITAL   (Japan)

^b OSAKA UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

C REACTIVE PROTEIN; GRANULOCYTE COLONY STIMULATING FACTOR; INTERLEUKIN 6;

CELL COUNT; CELL MOTION; HUMAN; INFLAMMATION; LETTER; LIVER CELL; LIVER FUNCTION; NEUTROPHIL; PRIORITY JOURNAL; PROTEIN BLOOD LEVEL;

C-REACTIVE PROTEIN; CHEMOTAXIS, LEUKOCYTE; CYTOPLASMIC GRANULES; HUMANS; INFLAMMATION; NEUTROPHILS;

EID: 0036092381     PISSN: 00029343     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0002-9343(02)91026-0     Document Type: Letter

References (5)

1. Toxic granulation neutrophils and C-reactive protein
2. Tumor necrosis factor (TNF) inhibits interleukin (IL)-1 and/or IL-6 stimulated synthesis of C-reactive protein (CRP) and serum amyloid A (SAA) in primary cultures of human hepatocytes
3. Correlation between serum IL-6 and CRP levels and severity of head injury in children
4. Studies of peripheral blood toxic neutrophils as a predictor of coronary risk in Kawasaki disease: The pathogenetic role of hematopoietic colony stimulating-factors (GM-CSF, G-CSF)
5. Serum cytokine levels (IL-4, IL-6, IL-8, G-CSF, GM-CSF) in burned patients