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Volumn 12, Issue 6, 2002, Pages 969-975
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A bioinformatics-based strategy identifies c-Myc and Cdc25A as candidates for the Apmt mammary tumor latency modifiers
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Author keywords
[No Author keywords available]
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Indexed keywords
MYC PROTEIN;
TRANSCRIPTION FACTOR;
VIRUS MIDDLE T ANTIGEN;
3' UNTRANSLATED REGION;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
BIOINFORMATICS;
BREAST TUMOR;
CARCINOGENESIS;
CELL CYCLE;
CONGENIC STRAIN;
CONTROLLED STUDY;
GENE CONSTRUCT;
GENE FUNCTION;
GENE IDENTIFICATION;
GENE INTERACTION;
GENE LOCUS;
GENE SEQUENCE;
GENETIC ANALYSIS;
GENETIC CODE;
GENETIC EPISTASIS;
GENETIC POLYMORPHISM;
GROWTH ACCELERATION;
LATENT PERIOD;
MOLECULAR MIMICRY;
MOLECULAR MODEL;
MOUSE;
NONHUMAN;
ONCOGENE C MYC;
ONSET AGE;
PHENOTYPE;
POLYOMA VIRUS;
PRIORITY JOURNAL;
PROMOTER REGION;
QUANTITATIVE TRAIT LOCUS MAPPING;
SEQUENCE ANALYSIS;
TRANSGENE;
3T3 CELLS;
ANIMALS;
CDC25 PHOSPHATASE;
COMPUTATIONAL BIOLOGY;
EPISTASIS, GENETIC;
GENES, MYC;
GENETIC MARKERS;
HUMANS;
MAMMARY NEOPLASMS, EXPERIMENTAL;
MICE;
MICE, TRANSGENIC;
PROTO-ONCOGENE PROTEINS C-MYC;
QUANTITATIVE TRAIT, HERITABLE;
ANIMALIA;
POLYOMAVIRUS;
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EID: 0036079155
PISSN: 10889051
EISSN: None
Source Type: Journal
DOI: 10.1101/gr.210502 Document Type: Article |
Times cited : (28)
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References (33)
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