The GSTM1 and GSTT1 genetic polymorphisms and susceptibility to acute lymphoblastic leukemia in children from north Portugal [3]

Leukemia

Volumn 16, Issue 8, 2002, Pages 1565-1567

  Alves, S ^a   Amorim, A ^a   Ferreira, F ^b   Norton, L ^b   Prata, M J ^a  

^a UNIVERSITY OF PORTO   (Portugal)

^b Hospital Geral   (Portugal)

Author keywords

[No Author keywords available]

Indexed keywords

GLUTATHIONE TRANSFERASE;

ACUTE LYMPHOBLASTIC LEUKEMIA; ALLELE; CHILD; CHILDHOOD LEUKEMIA; CLINICAL ARTICLE; CONTROLLED STUDY; FEMALE; GENE FREQUENCY; GENE MUTATION; GENETIC ASSOCIATION; GENETIC POLYMORPHISM; GENETIC RISK; GENETIC SUSCEPTIBILITY; GENOTYPE PHENOTYPE CORRELATION; HETEROZYGOSITY; HOMOZYGOSITY; HUMAN; LETTER; MALE; POLYMERASE CHAIN REACTION; POPULATION GENETICS; PORTUGAL; PRIORITY JOURNAL;

EID: 0036052581     PISSN: 08876924     EISSN: None     Source Type: Journal    
DOI: 10.1038/sj.leu.2402543     Document Type: Letter

References (8)

1. Genetic susceptibility to childhood acute lymphoblastic leukemia
2. Higher frequency of glutathione S-transferase deletions in black children with acute lymphoblastic leukemia
3. Susceptibility to childhood acute lymphoblastic leukemia: Influence of CYP1A1, CYP2D6, GSTM1, and GSTT1 genetic polymorphisms
4. Detection of the GSTM1* 0 allele by long polymerase chain reaction
5. Characterization of the glutathione S-transferase GSTT1 deletion: Discrimination of all genotypes by polymerase chain reaction indicates a trimodular genotype-phenotype correlation
6. Mammalian class theta GST and differential susceptibility to carcinogens: A review
7. Genetic polymorphism of CYP2D6, GSTM1 and NAT2 and susceptibility to haematological neoplasias
8. Glutathione S-transferase enzyme expression in hematopoietic cell lines implies a differential protective role for T1 and A1 isoenzymes in erythroid and for M1 in lymphoid lineages