J Wave and ST Segment Elevation in the Inferior Leads a Latent Type of Variant Brugada Syndrome ?

Japanese Heart Journal

Volumn 43, Issue 1, 2002, Pages 55-60

  Sahara, Makoto ^a   Sagara, Kouichi ^a   Yamashita, Takeshi ^a   Abe, Tsuyoshi ^a   Kirigaya, Hajime ^a   Nakada, Misao ^a   Iinuma, Hiroyuki ^a   Fu, Long Tai ^a   Watanabe, Hiroshi ^a  

^a THE CARDIOVASCULAR INSTITUTE   (Japan)

Author keywords

Brugada syndrome; Idiopathic ventricular fibrillation; Inferior leads; J wave; Pilsicainide; Sodium channel blockers; Ventricular tachycardia

Indexed keywords

DRUG DERIVATIVE; LIDOCAINE; PILSICAINIDE; SODIUM CHANNEL BLOCKING AGENT;

ADULT; ARTICLE; CASE REPORT; DIFFERENTIAL DIAGNOSIS; ELECTROCARDIOGRAPHY; HEART BUNDLE BRANCH BLOCK; HEART VENTRICLE FIBRILLATION; HEART VENTRICLE TACHYCARDIA; HUMAN; MALE; SYNDROME;

ADULT; BUNDLE-BRANCH BLOCK; DIAGNOSIS, DIFFERENTIAL; ELECTROCARDIOGRAPHY; HUMANS; LIDOCAINE; MALE; SODIUM CHANNEL BLOCKERS; SYNDROME; TACHYCARDIA, VENTRICULAR; VENTRICULAR FIBRILLATION;

EID: 0036050324     PISSN: 00214868     EISSN: 1348673X     Source Type: Journal    
DOI: 10.1536/jhj.43.55     Document Type: Article

References (10)

1. Brugada, P., Brugada J. Right bundle branch block, persistent ST segment elevation and sudden cardiac death: A distinct clinical and electrocardiographic syndrome. J Am Coll Cardiol 1992; 20: 1391–1396.
2. Takagi, M., Aihara, N., Takaki, H., et al. Clinical characteristics of patients with spontaneous or inducible ventricular fibrillation without apparent heart disease presenting with J wave and ST segment elevation in inferior leads. J Cardiovasc Electrophysiol 2000; 11: 844–6.
3. Kalla, H., Yan GX, Marinchak R. Ventricular fibrillation in a patient with prominent, J., (Osborn) waves and ST segment elevation in the inferior electrocardiographic leads: A Brugada syndrome variant? J Cardiovasc Electrophysiol 2000; 11: 95–98.
4. Brugada, J., Brugada P. Further characterization of the syndrome of right bundle branch block, ST segment elevation, and sudden cardiac death. J Cardiovasc Electrophysiol 1997; 8: 325–331.
5. Miyazaki, T., Mitamura, H., Miyoshi, S., Soejima, K., Aizawa, Y., Ogawa S. Autonomic and antiarrhythmic drug modulation of ST segment elevation in patients with Brugada syndrome. J Am Coll Cardiol 1996; 27: 1061–1070.
6. Fujiki, A., Usui, M., Nagasawa, H., Mizumaki, K., Hayashi, H., Inoue H. ST segment elevation in the right precor-dial leads induced with class IC antiarrhythmic drugs: Insight into the mechanism of Brugada syndrome. J Cardiovasc Electrophysiol 1999; 10: 214–218.
7. Chen, Q., Kirsch, G. E., Zhang, D., et al. Genetic basis and molecular mechanism for idiopathic ventricular fibrillation. Nature 1998; 392: 293–296.
8. Makita, N., Shirai, N., Wang, D. W., et al. Cardiac Na+ channel dysfunction in Brugada syndrome is aggravated by beta (1)-subunit. Circulation 2000; 101: 54–60.
9. Priori, S. G., Schwartz, P. J., Napolitano, C., et al. A recessive variant of the Romano-Ward long-QT syndrome? Circulation 1998; 97: 2420–2450.
10. Kubota, T., Shimizu, W., Kamakura, S., Horie M. Hypokalemia-induced long QT syndrome underlain by a novel missense mutation in S4-S5 linker of KCNQ1. J Cardiovasc Electrophysiol 2000; 11: 1048–1054.