Injury to the skin induces p53- and Fas-dependent bystander keratinocyte apoptosis in vivo [7]

British Journal of Dermatology

Volumn 147, Issue 3, 2002, Pages 614-616

  Hayashi, S ^a   Matsushima, H ^a   Shimada, Shinji ^a  

^a UNIVERSITY OF YAMANASHI   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

ANIMAL MODEL; APOPTOSIS; BYSTANDER EFFECT; CONTROLLED STUDY; DNA DAMAGE; ELECTROPHORESIS; FEMALE; HUMAN; IN VIVO STUDY; KERATINOCYTE; LETTER; MOUSE; NICK END LABELING; NONHUMAN; PRECANCER; PRIORITY JOURNAL; SKIN DEFECT; SKIN INJURY; STAINING; ULTRAVIOLET IRRADIATION;

EID: 0036042725     PISSN: 00070963     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1365-2133.2002.48817.x     Document Type: Letter

References (9)

1. T cell-mediated Fas-induced keratinocyte apoptosis plays a key pathogenetic role in eczematous dermatitis
2. Cell death by apoptosis in epidermal biology
3. Cellular proofreading
4. Fas ligand: A sensor for DNA damage critical in skin cancer etiology
5. Consequences of cell death: Exposure to necrotic tumor cells, but not primary tissue cells or apoptotic cells, induces the maturation of immunostimulatory dendritic cells
6. Phagocytosis triggers macrophage release of Fas ligand and induces apoptosis of bystander leukocytes
7. Phagocytosis of nonapoptotic cells dying by caspase-independent mechanisms
8. Apoptosis: Phagocytic docking without shocking
9. Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis