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Volumn 9, Issue 3, 2002, Pages 206-212
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Chronic hypoxia attenuates nitric oxide-dependent hemodynamic responses to acute hypoxia
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Author keywords
Hemodynamics; Nitric oxide; Pulmonary vasoconstriction; Systemic vasodilation
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Indexed keywords
N(G) NITROARGININE METHYL ESTER;
NITRIC OXIDE;
NITRIC OXIDE SYNTHASE INHIBITOR;
ENZYME INHIBITOR;
VASODILATOR AGENT;
ADAPTATION;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTERIAL PRESSURE;
ARTICLE;
CONTROLLED STUDY;
DRUG EFFECT;
HEART OUTPUT;
HEART RIGHT VENTRICLE HYPERTROPHY;
HEART VENTRICLE SEPTUM;
HEART WEIGHT;
HEMODYNAMICS;
HYPOBARISM;
HYPOXIA;
LUNG ARTERY PRESSURE;
LUNG VASCULAR RESISTANCE;
LUNG VASOCONSTRICTION;
MALE;
NONHUMAN;
PATHOGENESIS;
PRIORITY JOURNAL;
PULMONARY HYPERTENSION;
RAT;
SYSTEMIC VASCULAR RESISTANCE;
VASODILATATION;
ANIMAL;
ANOXIA;
BLOOD PRESSURE;
HYPOTENSION;
METABOLISM;
PHYSIOLOGY;
SPRAGUE DAWLEY RAT;
ANIMAL;
ANOXIA;
BLOOD PRESSURE;
ENZYME INHIBITORS;
HEMODYNAMICS;
HYPERTENSION, PULMONARY;
HYPOTENSION;
MALE;
NG-NITROARGININE METHYL ESTER;
NITRIC OXIDE;
RATS;
RATS, SPRAGUE-DAWLEY;
SUPPORT, NON-U.S. GOV'T;
VASODILATOR AGENTS;
ANIMALS;
HEMODYNAMIC PROCESSES;
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EID: 0036017208
PISSN: 10217770
EISSN: 14230127
Source Type: Journal
DOI: 10.1159/000059421 Document Type: Article |
Times cited : (11)
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References (32)
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