Deletion of the copper transporter CaCCC2 reveals two distinct pathways for iron acquisition in Candida albicans

Molecular Microbiology

Volumn 44, Issue 6, 2002, Pages 1551-1560

  Weissman, Ziva ^a   Shemer, Revital ^a   Kornitzer, Daniel ^a  

^a TECHNION ISRAEL INSTITUTE OF TECHNOLOGY   (Israel)

Author keywords

[No Author keywords available]

Indexed keywords

ADENOSINE TRIPHOSPHATASE; COPPER; HEMIN; HEMOGLOBIN; IRON; PROTEIN; PROTEIN CACCC2; PROTEIN CACRP1; UNCLASSIFIED DRUG; CARRIER PROTEIN; CATION TRANSPORT PROTEIN; CCC2 PROTEIN, S CEREVISIAE; SACCHAROMYCES CEREVISIAE PROTEIN;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BIOGENESIS; CANDIDA ALBICANS; CONTROLLED STUDY; DETOXIFICATION; FUNGAL STRAIN; GOLGI COMPLEX; IRON TRANSPORT; MALE; MOUSE; MYCOSIS; NONHUMAN; NUCLEOTIDE SEQUENCE; PHENOTYPE; PRIORITY JOURNAL; REGULATORY MECHANISM; SACCHAROMYCES CEREVISIAE; ANIMAL; BAGG ALBINO MOUSE; DISEASE MODEL; ENZYMOLOGY; GENE DELETION; GENE EXPRESSION REGULATION; GENETIC COMPLEMENTATION; GENETICS; METABOLISM; MOLECULAR GENETICS; PATHOGENICITY;

ADENOSINETRIPHOSPHATASE; ANIMAL; CANDIDA ALBICANS; CATION TRANSPORT PROTEINS; COPPER; DISEASE MODELS, ANIMAL; GENE DELETION; GENE EXPRESSION REGULATION, FUNGAL; GENETIC COMPLEMENTATION TEST; IRON; MEMBRANE TRANSPORT PROTEINS; MICE; MICE, INBRED BALB C; MOLECULAR SEQUENCE DATA; PHENOTYPE; SACCHAROMYCES CEREVISIAE; SACCHAROMYCES CEREVISIAE PROTEINS; SUPPORT, NON-U.S. GOV'T; ADENOSINE TRIPHOSPHATASES; ANIMALS;

ANIMALIA; CANDIDA; CANDIDA ALBICANS; SACCHAROMYCES; SACCHAROMYCES CEREVISIAE;

EID: 0036016820     PISSN: 0950382X     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1365-2958.2002.02976.x     Document Type: Article

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