No 123I-BMIPP accumulation in the myocardium and type I CD36 deficiency in a patient with acute subendocardial infarction: A case report

Kakuigaku

Volumn 39, Issue 1, 2002, Pages 29-35

  Asano, Yuuji ^a   Ishii, Katsumi ^a   Sagiuchi, Takao ^a   Tokita, Naoki ^a   Aoki, Yuki ^a   Woodhams, Reiko ^a   Katsunuma, Eita ^a   Izumi, Tohru ^a   Hayakawa, Kazushige ^a  

^a KITASATO UNIVERSITY SCHOOL OF MEDICINE   (Japan)

Author keywords

123I methyl p iodophenylpentadecanoic acid (123I BMIPP); Myocardial scintigraphy; No accumulation; Subendocardial infarction; Type I CD36 deficiency

Indexed keywords

15 (4 IODOPHENYL) 3 METHYLPENTADECANOIC ACID I 123; CD36 ANTIGEN; TETROFOSMIN TC 99M; (3 IODOBENZYL)GUANIDINE; DIAGNOSTIC AGENT; FATTY ACID; IODOBENZENE; IODOFILTIC ACID; ORGANOPHOSPHORUS COMPOUND; RADIOACTIVE IODINE; RADIOPHARMACEUTICAL AGENT; TECHNETIUM COMPLEX; TECHNETIUM TC 99M 1,2 BIS(BIS(2 ETHOXYETHYL)PHOSPHINO)ETHANE; TECHNETIUM TC 99M 1,2-BIS(BIS(2-ETHOXYETHYL)PHOSPHINO)ETHANE;

ACUTE SUBENDOCARDIAL INFARCTION; AGED; ARTICLE; CASE REPORT; DIAGNOSTIC IMAGING; HEART INFARCTION; HEART LEFT VENTRICLE FUNCTION; HEART MUSCLE; HEART SCINTISCANNING; HUMAN; MALE; MONOCYTE; PROTEIN DEFICIENCY; THROMBOCYTE; TYPE 1 CD36 DEFICIENCY; BLOOD; ENDOCARDIUM; HEART; METABOLISM; SCINTISCANNING;

3-IODOBENZYLGUANIDINE; AGED; ANTIGENS, CD36; ENDOCARDIUM; FATTY ACIDS; HEART; HUMANS; IODINE RADIOISOTOPES; IODOBENZENES; MALE; MYOCARDIAL INFARCTION; MYOCARDIUM; ORGANOPHOSPHORUS COMPOUNDS; ORGANOTECHNETIUM COMPOUNDS; RADIOPHARMACEUTICALS;

EID: 0036010262     PISSN: 00227854     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (13)

1. Metabolism substrate with negative myocardial uptake of iodine-123-BMIPP
2. Myocardial CD36 expression and fatty acid accumulation in patients with type I and II CD36 deficiency
3. 123I-BMIPP after 4 years
4. Improvement in cardiac function and free fatty acid metabolism in a case of dilated cardiomyopathy with CD36 deficiency
5. A platelet membrane glycoprotein deficiency in healthy blood donors
6. A novel polymorphism in glycoprotein IV (replacement of proline-90 by serine) predominates in subjects with platelet GPIV
7. Identification of molecular defects in a subject with type I CD36 deficiency
8. Molecular basis of CD36 deficiency - Evidence that a 478C-T substitution (proline90-serine) in CD36 cDNA account for CD36 deficiency