The channel hypothesis of Alzheimer's disease: Current status

Peptides

Volumn 23, Issue 7, 2002, Pages 1311-1315

  Kagan, Bruce L ^a   Hirakura, Yutaka ^b   Azimov, Rustam ^a   Azimova, Rushana ^a   Lin, Meng Chin ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b WASEDA UNIVERSITY   (Japan)

Author keywords

Amyloid; Apoptosis; Bilayers; Electrophysiology; Membranes; Pores

Indexed keywords

ALPHA TOXIN; ALUMINUM; AMYLOID BETA PROTEIN; AMYLOID BETA PROTEIN[1-40]; AMYLOID BETA PROTEIN[1-42]; CALCIUM ION; CESIUM ION; CHLORIDE ION; COLICIN; CONGO RED; DEFENSIN; LITHIUM ION; POTASSIUM CHANNEL; POTASSIUM ION; PROTEIN BAX; PROTEIN BCL 2; SODIUM ION; TROMETAMOL; UNCLASSIFIED DRUG; ZINC ION; ION CHANNEL; PEPTIDE FRAGMENT;

ALZHEIMER DISEASE; APOPTOSIS; ARTICLE; CALCIUM HOMEOSTASIS; CYTOTOXICITY; ELECTRIC POTENTIAL; HUMAN; LIPID BILAYER; MEMBRANE POTENTIAL; NEUROTOXICITY; NONHUMAN; PERMEABILITY; PRIORITY JOURNAL; ANIMAL; CELL MEMBRANE; CHEMISTRY; DRUG EFFECT; FORECASTING; METABOLISM; PHYSIOLOGY; REVIEW;

ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN; ANIMAL; CELL MEMBRANE; FORECASTING; HUMAN; ION CHANNELS; LIPID BILAYERS; PEPTIDE FRAGMENTS; SUPPORT, NON-U.S. GOV'T; SUPPORT, U.S. GOV'T, P.H.S.; ANIMALS; HUMANS;

EID: 0035997234     PISSN: 01969781     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0196-9781(02)00067-0     Document Type: Article

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