Effect of prolonged uremia on insulin-like growth factor-I receptor autophosphorylation and tyrosine kinase activity in kidney and muscle

Experimental Nephrology

Volumn 10, Issue 4, 2002, Pages 285-292

  Tsao, Tanny ^a   Fervenza, Fernando ^a   Friedlaender, Michael ^a   Chen, Yu ^a   Rabkin, Ralph ^a,b  

^a STANFORD UNIVERSITY   (United States)

^b VA PALO ALTO HEALTH CARE SYSTEM   (United States)

Author keywords

Acute renal failure; Chronic renal failure; Insulin like growth factor; Receptors; Signal transduction; Uremia

Indexed keywords

PROTEIN TYROSINE KINASE; SOMATOMEDIN C; SOMATOMEDIN C RECEPTOR; CREATININE;

ACUTE KIDNEY FAILURE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; AUTOPHOSPHORYLATION; CONTROLLED STUDY; DISEASE DURATION; DRUG RECEPTOR BINDING; ENZYME ACTIVITY; KIDNEY; MALE; MUSCLE; NONHUMAN; PRIORITY JOURNAL; PROTEIN FUNCTION; RAT; UREMIA; ANIMAL; BLOOD; BODY WEIGHT; CHRONIC KIDNEY FAILURE; METABOLISM; PHOSPHORYLATION; PHYSIOLOGY; SIGNAL TRANSDUCTION; SKELETAL MUSCLE; SOLUBILITY; SPRAGUE DAWLEY RAT;

ANIMALS; BODY WEIGHT; CREATININE; INSULIN-LIKE GROWTH FACTOR I; KIDNEY; KIDNEY FAILURE, ACUTE; KIDNEY FAILURE, CHRONIC; MALE; MUSCLE, SKELETAL; PHOSPHORYLATION; PROTEIN-TYROSINE KINASES; RATS; RATS, SPRAGUE-DAWLEY; RECEPTOR, IGF TYPE 1; SIGNAL TRANSDUCTION; SOLUBILITY; UREMIA;

EID: 0035991129     PISSN: 10187782     EISSN: None     Source Type: Journal    
DOI: 10.1159/000063703     Document Type: Article

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