ICP monitoring following bilateral carotid occlusion in GFAP-null mice

Acta Neurochirurgica, Supplement

Volumn 81, Issue , 2002, Pages 269-270

  Nawashiro, Hiroshi ^a   Huang, S ^a   Brenner, M ^b   Shima, K ^a   Hallenbeck, J M ^c  

^a NATIONAL DEFENSE MEDICAL COLLEGE   (Japan)

^b University of Alabama at Birmingham   (United States)

^c NATIONAL INSTITUTES OF HEALTH   (United States)

Author keywords

Cerebral ischemia; Glial fibrillary acidic protein; Intracranial pressure; Mice

Indexed keywords

GLIAL FIBRILLARY ACIDIC PROTEIN;

ANIMAL; ARTICLE; BLOOD BRAIN BARRIER; BLOOD FLOW VELOCITY; BRAIN CIRCULATION; CAROTID ARTERY OBSTRUCTION; DISEASE MODEL; GENETICS; HEMISPHERIC DOMINANCE; INTRACRANIAL PRESSURE; METHODOLOGY; MONITORING; MOUSE; MOUSE MUTANT; PATHOPHYSIOLOGY; PHYSIOLOGY;

ANIMALS; BLOOD FLOW VELOCITY; BLOOD-BRAIN BARRIER; CAROTID STENOSIS; CEREBROVASCULAR CIRCULATION; DISEASE MODELS, ANIMAL; FUNCTIONAL LATERALITY; GLIAL FIBRILLARY ACIDIC PROTEIN; INTRACRANIAL PRESSURE; MICE; MICE, KNOCKOUT; MONITORING, PHYSIOLOGIC;

EID: 0035989246     PISSN: 00651419     EISSN: None     Source Type: Journal    
DOI: 10.1007/978-3-7091-6738-0_69     Document Type: Article

References (4)

1. Altered taurine release following hypotonic stress in astrocytes from mice deficient for GFAP and vimentin
2. Brain microvessels: Factors altering their patency after the occlusion of a middle cerebral artery (Wistar Rat)
3. Targeted deletion in astrocyte intermediate filament (Gfap) alters neuronal physiology
4. High susceptibility to cerebral ischemia in GFAP-null mice