Metabolic suppressive therapy as a treatment for intracranial hypertension - Why it works and when it fails

Acta Neurochirurgica, Supplement

Volumn 81, Issue , 2002, Pages 69-70

  Oertel, M ^a   Kelly, Daniel F ^a   Lee, J H ^a   Glenn, T C ^a   Vespa, P M ^a   Martin, N A ^a  

^a UCLA MEDICAL CENTER   (United States)

Author keywords

Hyperglycolysis; ICP treatment; Metabolic suppressive therapy; Traumatic brain injury

Indexed keywords

ANTICONVULSIVE AGENT; BIOLOGICAL MARKER; CARBON DIOXIDE; OXYGEN; PROPOFOL;

ARTICLE; BLOOD; BRAIN CIRCULATION; BRAIN INJURY; HUMAN; INTRACRANIAL HYPERTENSION; PATHOPHYSIOLOGY; PHYSIOLOGY;

ANTICONVULSANTS; BIOLOGICAL MARKERS; BRAIN INJURIES; CARBON DIOXIDE; CEREBROVASCULAR CIRCULATION; HUMANS; INTRACRANIAL HYPERTENSION; OXYGEN; PROPOFOL;

EID: 0035989090     PISSN: 00651419     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (7)

1. Cerebral hyperglycolysis following severe traumatic brain injury in humans: A positron emission tomography study
2. Cerebral hemodynamic effects of pentobarbital coma in head-injured patients
3. Failure of high-dose steroid therapy to influence intracranial pressure in patients with severe head injury
4. The outcome with aggressive treatment in severe head injuries. Part II: Acute and chronic barbiturate administration in the management of head injury
5. Cerebral blood flow, vasoreactivity, and oxygen consumption during barbiturate therapy in severe traumatic brain lesions
6. Barbiturate-augmented hypothermia for reduction of persistent intracranial hypertension
7. Failure of prophylactic barbiturate coma in the treatment of severe head injury