Aldehyde dehydrogenase 3 gene regulation: Studies on constitutive and hypoxia-modulated expression

Chemico-Biological Interactions

Volumn 130-132, Issue , 2001, Pages 227-233

  Reisdorph, Richard ^a   Lindahl, Ronald ^a  

^a UNIVERSITY OF SOUTH DAKOTA   (United States)

Author keywords

Aldehyde dehydrogenase; ALDH3; Cornea; Gene regulation; Hepatoma cells; Hypoxia

Indexed keywords

ALDEHYDE DEHYDROGENASE; ALDEHYDE DEHYDROGENASE 3; AROMATIC HYDROCARBON RECEPTOR; CARRIER PROTEIN; HYPOXIA INDUCIBLE FACTOR 1; TRANSCRIPTION FACTOR; UNCLASSIFIED DRUG;

ANIMAL CELL; CONFERENCE PAPER; CONTROLLED STUDY; CORNEA CELL; ENZYME ACTIVATION; ENZYME INDUCTION; EPITHELIUM CELL; EXPRESSION VECTOR; GENE CONTROL; HYPOXIA; NONHUMAN; PROTEIN EXPRESSION; RAT; REPORTER GENE; XENOBIOTIC METABOLISM;

ALDEHYDE DEHYDROGENASE; ANIMALS; ANOXIA; ARYL HYDROCARBON RECEPTOR NUCLEAR TRANSLOCATOR; CELLS, CULTURED; CORNEA; DNA-BINDING PROTEINS; DOWN-REGULATION; EPITHELIAL CELLS; GENE EXPRESSION REGULATION, ENZYMOLOGIC; LIVER NEOPLASMS, EXPERIMENTAL; OXIDATIVE STRESS; RATS; RECEPTORS, ARYL HYDROCARBON; TRANSCRIPTION FACTORS; TUMOR CELLS, CULTURED; XENOBIOTICS;

EID: 0035969831     PISSN: 00092797     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0009-2797(00)00267-2     Document Type: Conference Paper

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4. Hypoxia exerts cell-type-specific effects on expression of the Class 3 aldehyde dehydrogenase gene
5. Isoform-specific induction of a human aldo-keto reductase by polycyclic aromatic hydrocarbons (PAH), electrophiles, and oxidative stress: Implications for the alternate pathway of PAH activation catalyzed by human dihydrodiol dehydrogenase
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