Are the complex regional pain syndromes due to neurogenic inflammation?

Neurology

Volumn 57, Issue 12, 2001, Pages 2161-2162

  Bennett, G J ^a  

^a MCGILL UNIVERSITY   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

EDITORIAL; HUMAN; NEUROGENIC INFLAMMATION; NOCICEPTION; PAIN; PATHOGENESIS; PRIORITY JOURNAL; SENSORY NERVE; VASODILATATION;

EID: 0035956517     PISSN: 00283878     EISSN: None     Source Type: Journal    
DOI: 10.1212/WNL.57.12.2161     Document Type: Editorial

References (14)

1. Die sogenannte acute knochenaptophie als entzündungsvorgang
2. Are toxic oxygen radicals involved in the pathogenesis of reflex sympathetic dystrophy?
3. Reflex sympathetic dystrophy of the hand: An excessive inflammatory response?
4. A novel animal model to evaluate oxygen derived free radical damage in soft tissue
5. Patterns of spread in complex regional pain syndrome, type I (reflex sympathetic dystrophy)
6. The significant role of neuropeptides in complex regional pain syndrome
7. Facilitated neurogenic inflammation in complex regional pain syndrome
8. Chronic hyperalgesia and skin warming caused by sensitized C nociceptors
9. Increased skin lactate in complex regional pain syndrome: Evidence for tissue hypoxia?
10. Effects of cutaneous histamine application in patients with sympathetic reflex dystrophy
11. Interactions between sympathetic vasoconstrictor outflow and C nociceptor-induced antidromic vasodilatation
12. Sudomotor function in sympathetic reflex dystrophy
13. Neuropathic pain
14. Despite clinical similarities there are significant differences between acute limb trauma and complex regional pain syndrome I (CRPS I)