Narcolepsy and low CSF orexin (hypocretin) concentration after a diencephalic stroke

Neurology

Volumn 56, Issue 12, 2001, Pages 1751-1753

  Scammell, T E ^a   Nishino, S ^b   Mignot, E ^b   Saper, C B ^a  

^a BETH ISRAEL DEACONESS MEDICAL CENTER   (United States)

^b STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

OREXIN;

ADULT; ARTICLE; CASE REPORT; HUMAN; HYPOTHALAMUS; MALE; NARCOLEPSY; NEUROPATHOLOGY; PRIORITY JOURNAL; PROTEIN CEREBROSPINAL FLUID LEVEL; STROKE;

EID: 0035954346     PISSN: 00283878     EISSN: None     Source Type: Journal    
DOI: 10.1212/WNL.56.12.1751     Document Type: Article

References (10)

1. Sleep as a problem of localization
2. Narcolepsy in orexin knockout mice: Molecular genetics of sleep regulation
3. The sleep disorder canine narcolepsy is caused by a mutation in the hypocretin (orexin) receptor 2 gene
4. A mutation in a case of early onset narcolepsy and a generalized absence of hypocretin peptides in human narcoleptic brains
5. Hypocretin (orexin) deficiency in human narcolepsy
6. Reduced number of hypocretin neurons in human narcolepsy
7. Diagnostic value of low CSF hypocretin/orexin levels in human narcolepsy
8. Neurons containing hypocretin (orexin) project to multiple neuronal systems
9. Symptomatic narcolepsies
10. Neuroimaging study in autosomal dominant cerebellar ataxia, deafness, and narcolepsy