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Volumn 1517, Issue 2, 2001, Pages 212-219
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TCF11/Nrf1 overexpression increases the intracellular glutathione level and can transactivate the γ-glutamylcysteine synthetase (GCS) heavy subunit promoter
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Author keywords
Glutamylcysteine synthetase; Antioxidant response element; Glutathione; TCF11 Nrf1
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Indexed keywords
GLUTAMATE CYSTEINE LIGASE;
GLUTATHIONE;
TRANSCRIPTION FACTOR;
TRANSCRIPTION FACTOR NRF1;
TRANSCRIPTION FACTOR NRF2;
TRANSCRIPTION FACTOR TCF11;
UNCLASSIFIED DRUG;
ANIMAL CELL;
ARTICLE;
CELL LEVEL;
CELL STRAIN COS1;
CONTROLLED STUDY;
DNA RESPONSIVE ELEMENT;
ENZYME SYNTHESIS;
GENE EXPRESSION REGULATION;
GLUTATHIONE METABOLISM;
NONHUMAN;
PRIORITY JOURNAL;
PROMOTER REGION;
TRANSACTIVATION;
AMINOACYLTRANSFERASES;
ANIMALS;
CLONING, MOLECULAR;
COS CELLS;
DIMERIZATION;
DNA-BINDING PROTEINS;
GENE EXPRESSION REGULATION, ENZYMOLOGIC;
GLUTATHIONE;
NF-E2-RELATED FACTOR 1;
PROMOTER REGIONS (GENETICS);
REPRESSOR PROTEINS;
TRANS-ACTIVATORS;
TRANSCRIPTION FACTORS;
TRANSFECTION;
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EID: 0035951489
PISSN: 01674781
EISSN: None
Source Type: Journal
DOI: 10.1016/S0167-4781(00)00276-1 Document Type: Article |
Times cited : (62)
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References (31)
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