Mechanisms of prion-induced modifications in membrane transport properties: Implications for signal transduction and neurotoxicity

Chemico-Biological Interactions

Volumn 138, Issue 1, 2001, Pages 1-26

  Kourie, Joseph I ^a  

^a AUSTRALIAN NATIONAL UNIVERSITY   (Australia)

Author keywords

Amyloidosis; Ion channel pathologies; Ion channel forming peptides; Membrane physiology; Neurodegenerative diseases; Prion diseases

Indexed keywords

4 AMINOBUTYRIC ACID; ADENOSINE TRIPHOSPHATASE; AMINO ACID; CALCINEURIN; CALCIUM CHANNEL L TYPE; CALCIUM ION; CALMODULIN; COPPER ION; DILTIAZEM; ION CHANNEL; ISOPROTEIN; MAGNESIUM ION; N METHYL DEXTRO ASPARTIC ACID; NIFEDIPINE; POLYPEPTIDE; PRION PROTEIN; SODIUM ION; SUPEROXIDE DISMUTASE; VERAPAMIL;

BETA SHEET; BINDING SITE; BIODIVERSITY; CELL MEMBRANE; CELL MEMBRANE FLUIDITY; CHANNEL GATING; CYTOTOXICITY; ELECTROPHYSIOLOGY; HOMEOSTASIS; HUMAN; ION TRANSPORT; MEMBRANE PERMEABILITY; MEMBRANE TRANSPORT; NEUROTOXICITY; NONHUMAN; PRION; PROTEIN AGGREGATION; PROTEIN INTERACTION; REDUCTION; REGULATORY MECHANISM; RIGIDITY; SCRAPIE; SHORT SURVEY; SIGNAL TRANSDUCTION; VISCOSITY;

ANIMALS; BRAIN; CATTLE; CELL MEMBRANE; HUMANS; ION TRANSPORT; PEPTIDE FRAGMENTS; PRION DISEASES; PRIONS; SIGNAL TRANSDUCTION;

EID: 0035950434     PISSN: 00092797     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0009-2797(01)00228-9     Document Type: Short Survey

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