Birthweights, maternal cardiovascular events, and Barker hypothesis

Lancet

Volumn 357, Issue 9273, 2001, Pages 1990-1991

  Poulter, Neil R ^a  

^a IMPERIAL COLLEGE SCHOOL OF MEDICINE   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

ALCOHOL ABUSE; ANTHROPOMETRY; DELIVERY; DIABETES MELLITUS; DISEASE PREDISPOSITION; ENVIRONMENTAL FACTOR; GENOTYPE; HEART INFARCTION; HEREDITY; HUMAN; HYPERTENSION; ISCHEMIC HEART DISEASE; LOW BIRTH WEIGHT; MATERNAL CARE; MONOZYGOTIC TWINS; MORTALITY; NOTE; PREECLAMPSIA; PREMATURITY; PRIORITY JOURNAL; PROGENY; RISK FACTOR; SMOKING; UTERUS;

EID: 0035938918     PISSN: 01406736     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0140-6736(00)05131-X     Document Type: Note

References (9)

1. Increased systolic blood pressure in adult rats induced by fetal exposure to maternal low protein diets
2. Early origin of coronary heart disease (the "Barker hypothesis")
3. Ordeals for the fetal programming hypothesis
4. "Place of publication" bias?
5. The role of size at birth and postnatal catch-up growth in determining systolic blood pressure: A systematic review of the literature
6. The fetal insulin hypothesis: An alternative explanation of the association of low birthweight with diabetes and vascular disease
7. Birth dimensions of offspring, premature birth, and the mortality of mothers
8. Association between birthweight and adult blood pressures of twins: A historical cohort study