Epidermal growth factor receptor signaling and response of cancer cells to ionizing radiation

Journal of the National Cancer Institute

Volumn 93, Issue 12, 2001, Pages 890-891

  Ethier, Stephen P ^a   Lawrence, Theodore S ^a,b  

^a UNIVERSITY OF MICHIGAN   (United States)

^b UNIVERSITY OF MICHIGAN MEDICAL SCHOOL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CETUXIMAB; EPIDERMAL GROWTH FACTOR RECEPTOR; MITOGEN ACTIVATED PROTEIN KINASE; PHOSPHOTRANSFERASE INHIBITOR; VIRUS VECTOR;

ADENOVIRUS; BREAST CANCER; CANCER CELL; CANCER INHIBITION; CELL PROLIFERATION; EDITORIAL; GENE THERAPY; HUMAN; HUMAN CELL; IMAGING; IONIZING RADIATION; PHENOTYPE; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; RADIATION DOSE FRACTIONATION; RADIOSENSITIZATION; SIGNAL TRANSDUCTION; TREATMENT OUTCOME; TUMOR BIOPSY; TUMOR XENOGRAFT;

EID: 0035918847     PISSN: 00278874     EISSN: None     Source Type: Journal    
DOI: 10.1093/jnci/93.12.890     Document Type: Editorial

References (7)

1. The EGF receptor family as targets for cancer therapy
2. Epidermal growth factor receptor as a genetic therapy target for carcinoma cell radiosensitization
3. Radiation-induced proliferation of the human A431 squamous carcinoma cells is dependent on EGFR tyrosine phosphorylation
4. Inhibition of the mitogen activated protein (MAP) kinase cascade potentiates cell killing by low dose ionizing radiation in A431 human squamous carcinoma cells
5. The inducible expression of dominant-negative epidermal growth factor receptor-CD533 results in radiosensitization of human mammary carcinoma cells
6. Inhibitory effects of combinations of HER-2/neu antibody and chemotherapeutic agents used for treatment of human breast cancers
7. r MoAb combined with RT in advanced head and neck malignancies