Isoforms of protein kinase C in postsynaptic densities after cerebral ischemia

Brain Research

Volumn 889, Issue 1-2, 2001, Pages 105-111

  Zablocka, Barbara ^a   Gajkowska, Barbara ^a   Czechmańska, Teresa ^a   Domańska Janik, Krystyna ^a  

^a MOSSAKOWSKI MEDICAL RESEARCH CENTRE   (Poland)

Author keywords

Brain ischemia; Isoforms of protein kinase C; Postsynaptic density; Synapse morphology

Indexed keywords

CALCIUM ION; ISOENZYME; PROTEIN KINASE C;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN CORTEX; BRAIN ISCHEMIA; CONTROLLED STUDY; ENZYME LOCALIZATION; GERBIL; HIPPOCAMPUS; IMMUNOCYTOCHEMISTRY; MALE; NONHUMAN; POLYACRYLAMIDE GEL ELECTROPHORESIS; POSTSYNAPTIC MEMBRANE; PRIORITY JOURNAL; PROTEIN ANALYSIS; RAT; REPERFUSION; SYNAPTIC TRANSMISSION; TRANSIENT ISCHEMIC ATTACK;

ANIMALS; BLOTTING, WESTERN; BRAIN ISCHEMIA; DENSITOMETRY; ELECTROPHORESIS, POLYACRYLAMIDE GEL; GERBILLINAE; IMMUNOBLOTTING; IMMUNOHISTOCHEMISTRY; ISOENZYMES; MALE; MICROSCOPY, ELECTRON; NERVE TISSUE PROTEINS; PROTEIN KINASE C; RATS; RATS, WISTAR; SUBCELLULAR FRACTIONS; SYNAPSES;

EID: 0035910352     PISSN: 00068993     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0006-8993(00)03122-X     Document Type: Article

References (28)

1. Time course of the translocation and inhibition of protein kinase C during complete cerebral ischemia in the rat
2. The structure of postsynaptic densities isolated from dog cerebral cortex. I. Overall morphology and protein composition
3. Protein kinase C as an early and sensitive marker of ischemia-induced progressive neuronal damage in gerbil hippocampus
4. Phosphorylation of protein kinase C substrate proteins in rat hippocampal slices - Effect of calpain inhibitors
5. Ischemia-induced posttranslational modifications of protein component of a rat brain post-synaptic densities (PSD) preparation
6. Effect of brain ischemia on protein kinase C
7. Evidence that the early loss of membrane protein kinase C is a necessary step in the excitatory amino acid-induced death of primary cortical neurons
8. Clustering membrane proteins: It's all coming together with the PSD-95/SAP90 protein family
9. Assembly of proteins to postsynaptic densities after transient cerebral ischemia
10. 2+/calmodulin-dependent protein kinase II to synaptic junctions in the vulnerable hippocamapl CA1 region following transient ischemia
11. + channels by direct interaction with the PSD95/SAP90 family of membrane-associated guanylate kinases
12. Delaued neuronal death in the gerbil hippocampus followed ischemia
13. Coordination of three signaling enzymes by AKAP 79, a mammalian scaffold protein
14. Domain interaction between NMDA receptor subunits and the postsynaptic density protein PSD-95
15. Protein measurement with the Folin phenol reagent
16. Modification of postsynaptic densities after transient cerebral ischemia: A quantitative and three-dimensional ultrastructural study
17. Localization of protein kinases by anchoring proteins: A theme in signal Transduction
18. Regulation of protein kinase C
19. Activation of novel protein kinases Cδ and ε upon mitogenic stimulation of quiescent rat 3Y1 fibroblasts
20. Identification and localization of an actin-binding motif that is unique to the epsilon isoform of protein kinase C and participates in the regulation of synaptic function
21. PDZs and receptor/channel clustering: Rounding up the latest suspects
22. Brain-derved neurotrophic factor rapidly enhances phosphorylation of the postsynaptic N-methyl-D-aspartate receptor subunit 1
23. NMDA receptor subunits in the postsynaptic density of rat brain: Expression and phosphorylation by endogenous protein kinases
24. Characterization of protein kinse C activities in postsynaptic density fraction prepared from cerebral cortex, hippocampus, and cerebellum
25. Evidence that brain-derived neurotrophic factor neuroprotection is linked to its ability to reverse the NMDA-induced inactivation of protein kinase C in cortical neurons
26. A multivalent PDZ-domain protein assembles signalling complexes in a G-protein-coupled cascade
27. Measurement of hippocampal levels of cellular second messengers following in situ freezing
28. Cell signaling and ischemic neuronal death