Tsp509I polymorphism in exon 2 of the glucocorticoid receptor gene in relation to obesity and cortisol secretion: Cohort study

British Medical Journal

Volumn 322, Issue 7287, 2001, Pages 652-653

  Rosmond, R ^a   Bouchard, C ^a   Bjorntorp P ^b  

^a PENNINGTON BIOMEDICAL RESEARCH CENTER   (United States)

^b SAHLGRENSKA UNIVERSITY HOSPITAL   (Sweden)

Author keywords

[No Author keywords available]

Indexed keywords

CHOLESTEROL; DEXAMETHASONE; GLUCOSE; HYDROCORTISONE; INSULIN; LEPTIN; TRIACYLGLYCEROL;

ADULT; ARTICLE; BODY FAT; CLINICAL TRIAL; CONTROLLED CLINICAL TRIAL; CONTROLLED STUDY; EXON; GENE ISOLATION; GENETIC POLYMORPHISM; GENOTYPE; HUMAN; HUMAN CELL; HUMAN TISSUE; HYDROCORTISONE RELEASE; MAJOR CLINICAL STUDY; MALE; OBESITY; PRIORITY JOURNAL; RANDOMIZED CONTROLLED TRIAL; RECEPTOR GENE;

EID: 0035901268     PISSN: 09598146     EISSN: None     Source Type: Journal    
DOI: 10.1136/bmj.322.7287.652     Document Type: Article

References (5)

1. High penetrance, overweight, and glucocorticoid receptor variant: Case-control study
2. A polymorphism in the glucocorticoid receptor gene may be associated with an increased sensitivity to glucocordcoids in vivo
3. Stress-related cortisol secretion in men: Relationships with abdominal obesity and endocrine, metabolic and hemodynamic abnormalities
4. Lack of association between five polymorphisms in the human glucocorticoid receptor gene and glucocorticoid resistance
5. A glucocorticoid receptor gene marker is associated with abdominal obesity, leptin, and dysregulation of the hypothalamic-pituitary-adrenal axis