Identification and sequencing the juvenile spermatogonial depletion critical interval on mouse chromosome 1 reveals the presence of eight candidate genes

Biochemical and Biophysical Research Communications

Volumn 288, Issue 5, 2001, Pages 1129-1135

  Boettger Tong, Holly L ^a   Rohozinski, Jan ^a   Agoulnik, Alexander I ^a   Dohmae, Kayoko ^c   Nishimune, Yoshitake ^c   Levy, Nicholas ^b   Bishop, Colin E ^a  

^a BAYLOR COLLEGE OF MEDICINE   (United States)

^b INSERM   (France)

^c OSAKA UNIVERSITY   (Japan)

Author keywords

jsd mutation; Linkage; Physical map; Spermatogenesis; Testis

Indexed keywords

ANIMAL TISSUE; ARTICLE; CELL DIFFERENTIATION; CHROMOSOME 1; FEMALE; GENE LOCUS; GENE MAPPING; GENE MUTATION; GENE SEQUENCE; MALE; MALE STERILITY; MOUSE; NONHUMAN; NUCLEOTIDE SEQUENCE; PRIORITY JOURNAL; SPERMATOGENESIS; SPERMATOGONIUM;

EID: 0035900619     PISSN: 0006291X     EISSN: None     Source Type: Journal    
DOI: 10.1006/bbrc.2001.5899     Document Type: Article

References (17)

1. MiRP1 forms IKr potassium channels with HERG and is associated with cardiac arrhythmia
2. Juvenile spermatogonial depletion (jsd): A genetic defect of germ cell proliferation of male mice
3. Juvenile spermatogonial depletion (jsd) mutant seminiferous tubules are capable of supporting transplanted spermatogenesis
4. Protease nexin I (PN1) in mouse brain is expressed from the same gene as in seminal vesicle
5. Stem cells in the testis
6. Arrest of spermatogonial differentiation in jsd/jsd, Sl17H/Sl17H, and cryptorchid mice
7. The Sp4H deletion may contain a new locus essential for postimplantation development
8. Pax-3, a novel murine DNA binding protein expressed during early neurogenesis
9. Sexually dimorphic expression of protease nexin-1 and vanin-1 in the developing mouse gonad prior to overt differentiation suggests a role in mammalian sexual development
10. Variable and multiple expression of protease nexin-1 during mouse organogenesis and nervous system development
11. Loss of sperm in juvenile spermatogonial depletion (jsd) mutant mice is ascribed to a defect of intratubular environment to support germ cell differentiation
12. Male fertility defects in mice lacking the serine protease inhibitor protease nexin-1
13. Defect in germ cells, not in supporting cells, is the cause of male infertility in the jsd mutant mouse: Proliferation of spermatogonial stem cells without differentiation
14. Transcriptional checkpoints determining the fate of male germ cells
15. The organisation of the mouse secretogranin II gene
16. Cullin-3 targets cyclin E for ubiquitination and controls S phase in mammalian cells
17. Cloning of the cDNA encoding phenylalanyl tRNA synthetase regulatory alpha-subunit-like protein whose expression is down-regulated during differentiation