The mouse Hoxd13spdh mutation, a polyalanine expansion similar to human type II synpolydactyly (SPD), disrupts the function but not the expression of other Hoxd genes

Developmental Biology

Volumn 237, Issue 2, 2001, Pages 345-353

  Bruneau, Sylvia ^a   Johnson, Kenneth R ^b   Yamamoto, Masakazu ^c   Kuroiwa, Atsushi ^c   Duboule, Denis ^a  

^a UNIVERSITY OF GENEVA   (Switzerland)

^b JACKSON LABORATORY   (United States)

^c NAGOYA UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

ALANINE; GENE PRODUCT; PROTEIN HOXA3 SPDH; PROTEIN HOXD; UNCLASSIFIED DRUG;

ALLELE; ANIMAL CELL; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; GENE DISRUPTION; GENE DUPLICATION; GENE EXPRESSION; GENE FUNCTION; GENE INDUCTION; GENE MUTATION; GROWTH RETARDATION; HOMOZYGOSITY; LIMB MALFORMATION; MARKER GENE; MORPHOLOGY; MOUSE; NONHUMAN; OSSIFICATION; PHENOTYPE; POLYDACTYLY; PRIORITY JOURNAL; PROTEIN INDUCTION; SEQUENCE HOMOLOGY; SYNDACTYLY;

EID: 0035884587     PISSN: 00121606     EISSN: None     Source Type: Journal    
DOI: 10.1006/dbio.2001.0382     Document Type: Article

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