Antitissue transglutaminase antibodies in HIV infection and effect of highly active antiretroviral therapy [2]

Journal of Acquired Immune Deficiency Syndromes

Volumn 27, Issue 5, 2001, Pages 507-508

  Pereda, Iliana ^a   Bartolomé Pacheco, María J ^a   Martín, Margarita ^a   López Escribano, Herminio ^a   Echevarría, Santiago ^a   López Hoyos, Marcos ^a  

^a HOSPITAL UNIVERSITARIO MARQUÉS DE VALDECILLA   (Spain)

Author keywords

[No Author keywords available]

Indexed keywords

ANTIBODY; ANTIRETROVIRUS AGENT; CARDIOLIPIN ANTIBODY; PROTEIN GLUTAMINE GAMMA GLUTAMYLTRANSFERASE;

ANTIBODY DETECTION; ANTIBODY PRODUCTION; ANTIBODY TITER; APOPTOSIS; DRUG EFFECT; HUMAN IMMUNODEFICIENCY VIRUS INFECTION; IMMUNE RESPONSE; LETTER; PRIORITY JOURNAL; VIRUS LOAD;

EID: 0035881007     PISSN: 15254135     EISSN: None     Source Type: Journal    
DOI: 10.1097/00126334-200108150-00015     Document Type: Letter

References (7)

1. Anti-phospholipid antibodies and CD5+ B cells in HIV infection
2. Autoantigens as substrates for apoptotic proteases: Implications for the pathogenesis of systemic autoimmune disease
3. Induction of "tissue" transglutaminase in HIV pathogenesis: Evidence for high rate of apoptosis of CD4+ T lymphocytes and accessory cells in lymphoid tissues
4. Anticardiolipin antibodies and dependence of a serum cofactor. A mechanism of thrombosis
5. Identification of tissue transglutaminase as the autoantigen of celiac disease
6. Programmed cell death as a mechanism of CD4 and CD8 T cell deletion in AIDS. Molecular control and effect of highly active anti-retroviral therapy
7. Antiphospholipid antibodies are an epiphenomenon in HIV-infected patients