Discrepancy between phenotype and genotype on screening for factor V Leiden after transplantation [2]

Blood

Volumn 97, Issue 8, 2001, Pages 2525-2526

  Parker, J ^a   Pagliuca, A ^a   Kitiyakara, T ^a   Whitehead, M ^a   Heaton, N ^a   O'Grady, J ^a   Arya, R ^a  

^a KING S COLLEGE HOSPITAL   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

ACTIVATED PROTEIN C; BLOOD CLOTTING FACTOR 5 LEIDEN;

ACTIVATED PROTEIN C RESISTANCE; ADULT; CASE REPORT; FEMALE; GENE MUTATION; GENETIC SCREENING; GENOTYPE; HETEROZYGOSITY; HUMAN; LETTER; LIVER TRANSPLANTATION; PHENOTYPE; PRIORITY JOURNAL; RISK FACTOR; VEIN THROMBOSIS;

EID: 0035871678     PISSN: 00064971     EISSN: None     Source Type: Journal    
DOI: 10.1182/blood.V97.8.2525     Document Type: Letter

References (9)

1. High risk of thrombosis in patients homozygous for factor V Leiden (activated protein C resistance)
2. Mutation in blood coagulation factor V associated with resistance to activated protein C
3. Familial thrombophilia due to a previously unrecognized mechanism characterized by poor anticoagulant response to activated protein C: Prediction of a cofactor to activated protein C
4. Resistance to activated protein C and factor V Leiden as risk factors for venous thrombosis
5. 306→Thr) associated with resistance to activated protein C
6. Risk of venous thromboembolism in users of hormone replacement therapy
7. Modified test for activated protein C resistance
8. Secretable human platelet-derived factor V originates from the plasma pool
9. Biosynthesis of coagulation factor V by a human hepatocellular carcinoma cell line