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Volumn 71, Issue 7, 2001, Pages 986-992

Clinical and biochemical outcome of hepatorenal transplantation for hereditary systemic amyloidosis associated with apolipoprotein AI Gly26Arg

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID PRECURSOR PROTEIN; AMYLOID PROTEIN; APOLIPOPROTEIN A1; ARGININE; GLYCINE;

EID: 0035870770     PISSN: 00411337     EISSN: None     Source Type: Journal    
DOI: 10.1097/00007890-200104150-00026     Document Type: Article
Times cited : (60)

References (39)
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    • (1991) , pp. 385
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  • 9
    • 0032887169 scopus 로고    scopus 로고
    • 174 ->Ser causes hereditary cardiac amyloidosis, and the amyloid fibrils are constituted by the 93-residue N-terminal polypeptide
    • (1999) Am J Pathol , vol.155 , Issue.3 , pp. 695
    • Obici, L.1    Bellotti, V.2    Mangione, P.3
  • 18
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    • Studies with radiolabelled serum amyloid P component provide evidence for turnover and regression of amyloid deposits in vivo
    • (1994) Clin Sci (Colch) , vol.87 , pp. 289
    • Hawkins, P.N.1
  • 33
    • 0031960611 scopus 로고    scopus 로고
    • The results of combined liver/kidney transplantation for primary hyperoxaluria (PH1) 1984-1997. The European PH1 transplant registry report
    • European PH1 Transplantation Study Group
    • (1998) J Nephrol , vol.11 , Issue.SUPPL. 1 , pp. 36
    • Jamieson, N.V.1
  • 38
    • 0034020609 scopus 로고    scopus 로고
    • Curative hepatorenal transplantation in systemic amyloidosis caused by the Glu526Val fibrinogen α-chain variant in an English family
    • (2000) QJM , vol.93 , pp. 269
    • Gillmore, J.D.1    Booth, D.R.2    Rela, M.3


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.