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Volumn 287, Issue 1, 2001, Pages 66-70

Oral exposure of dimethylarsinic acid, a main metabolite of inorganic arsenics, in mice leads to an increase in 8-oxo-2′-deoxyguanosine level, specifically in the target organs for arsenic carcinogenesis

Author keywords

8 oxo 2 deoxyguanosine (8 oxodG); Arsenic; Carcinogenesis; Dimethylarsinic acid; Free radical

Indexed keywords

8 HYDROXYDEOXYGUANOSINE; ARSENIC; BIOLOGICAL MARKER; CACODYLIC ACID; DNA; FREE RADICAL;

EID: 0035860357     PISSN: 0006291X     EISSN: None     Source Type: Journal    
DOI: 10.1006/bbrc.2001.5551     Document Type: Article
Times cited : (95)

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    • Wei, H.1    Frenkel, K.2
  • 24
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    • Recent advances in arsenic carcinogenesis: Modes of action, animal model systems, and methylated arsenic metabolites
    • (2001) Toxicol. Appl. Pharmacol. , vol.172 , pp. 249-261
    • Kitchin, K.T.1


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.