Rate of cognitive decline in AD is accelerated by the interleukin-1α -889*1 allele

Neurology

Volumn 56, Issue 11, 2001, Pages 1595-1597

  Murphy Jr , Greer M ^a,b   Claassen, J D ^a   DeVoss, J J ^a   Pascoe, N ^a,b   Taylor, J ^a,b   Tinklenberg, J R ^a,b   Yesavage, J A ^a,b  

^a STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b VETERANS AFFAIRS MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

INTERLEUKIN 1ALPHA;

ALZHEIMER DISEASE; ARTICLE; COGNITION; CONTROLLED STUDY; FEMALE; GENE FREQUENCY; GENETIC POLYMORPHISM; GENOTYPE; HOMOZYGOSITY; HUMAN; MAJOR CLINICAL STUDY; MALE; PRIORITY JOURNAL; RISK ASSESSMENT; RISK FACTOR;

EID: 0035849525     PISSN: 00283878     EISSN: None     Source Type: Journal    
DOI: 10.1212/WNL.56.11.1595     Document Type: Article

References (10)

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4. An IL-1 alpha susceptibility polymorphism in Alzheimer's disease: New fuel for the inflammation hypothesis
5. Association of an interleukin 1 alpha polymorphism with Alzheimer's disease
6. Association of early-onset Alzheimer's disease with an interleukin-1 alpha gene polymorphism
7. Association of interleukin-1 gene polymorphisms with Alzheimer's disease
8. A genetic association between juvenile rheumatoid arthritis and a novel interleukin-1 alpha polymorphism
9. Immunization with amyloid-beta attenuates Alzheimer-disease-like pathology in the PDAPP mouse
10. Interleukin-1 beta activates forebrain glial cells and increases nitric oxide production and cortical glutamate and GABA release in vivo: Implications for Alzheimer's disease