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Volumn 897, Issue 1-2, 2001, Pages 104-113
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Increased extracellular ascorbate release reflects glutamate re-uptake during the early stage of reperfusion after forebrain ischemia in rats
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Author keywords
Antioxidant; Ascorbic acid; Glutamate; In vivo electrochemistry; Ischemica reperfusion; Microdialysis biosensor
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Indexed keywords
ASCORBIC ACID;
GLUTAMATE OXIDASE;
GLUTAMIC ACID;
OXIDOREDUCTASE;
SODIUM CHLORIDE;
UNCLASSIFIED DRUG;
AMINO ACID TRANSPORT;
ANALYTIC METHOD;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ARTICLE;
BIOSENSOR;
BRAIN ISCHEMIA;
BRAIN LEVEL;
CAROTID ARTERY OBSTRUCTION;
CONTROLLED STUDY;
DISEASE DURATION;
ELECTRODE;
HEMORRHAGIC HYPOTENSION;
HIPPOCAMPUS;
MALE;
MEDIATOR RELEASE;
MICRODIALYSIS;
NONHUMAN;
PRIORITY JOURNAL;
RAT;
REPERFUSION;
ANIMALS;
ANTIOXIDANTS;
ASCORBIC ACID;
BIOSENSING TECHNIQUES;
EXTRACELLULAR SPACE;
GLUTAMIC ACID;
ISCHEMIC ATTACK, TRANSIENT;
MALE;
MICRODIALYSIS;
PROSENCEPHALON;
RATS;
RATS, WISTAR;
REPERFUSION INJURY;
TIME FACTORS;
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EID: 0035815492
PISSN: 00068993
EISSN: None
Source Type: Journal
DOI: 10.1016/S0006-8993(01)02099-6 Document Type: Article |
Times cited : (41)
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References (42)
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