Serum-activated assembly and membrane translocation of an endogenous Rac1: Effector complex

Current Biology

Volumn 11, Issue 5, 2001, Pages 356-360

  Hansen, Marc D H ^a   Nelson, W James ^a  

^a STANFORD UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANIMALIA; CANIS FAMILIARIS;

EID: 0035814886     PISSN: 09609822     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0960-9822(01)00091-4     Document Type: Article

References (20)

1. Stimulation of membrane ruffling and MAP kinase activation by distinct effectors of RAS
2. The cellular functions of small GTP-binding proteins
3. Rho: Theme and variations
4. Cell adhesion and Rho small GTPases
5. Sec6/8 complex is recruited to cell-cell contacts and specifies transport vesicle delivery to the basal-lateral membrane in epithelial cells
6. Rabbit intestine contains a protein that inhibits the dissociation of GDP from and the subsequent binding of GTP to rhoB p20, a ras p21-like GTP-binding protein
7. Molecular cloning and characterization of a novel type of regulatory protein (GDI) for the rho proteins, ras p21-like small GTP-binding proteins
8. A GDP dissociation inhibitor that serves as a GTPase inhibitor for the Ras-like protein CDC42Hs
9. A brain serine/ threonine protein kinase activated by Cdc42 and Rac1
10. Rho family GTPases regulate p38 mitogen-activated protein kinase through the downstream mediator Pak1
11. Human p21-activated kinase (Pak1) regulates actin organization in mammalian cells
12. Expression of constitutively active α-PAK reveals effects of the kinase on actin and focal complexes
13. Kinase-deficient Pak1 mutants inhibit Ras transformation of Rat-1 fibroblasts
14. Ankyrin binding to (Na+ + K+)ATPase and implications for the organization of membrane domains in polarized cells
15. Dominant inhibitory Ras mutants selectively inhibit the activity of either cellular or oncogenic Ras
16. Dominant inhibitory mutations in the Mg(2+)-binding site of Rash prevent its activation by GTP
17. Effects of regulated expression of mutant RhoA and Rac1 small GTPases on the development of epithelial (MDCK) cell polarity
18. Localization of p21-activated kinase 1 (PAK1) to pinocytic vesicles and cortical actin structures in stimulated cells
19. C-terminal binding domain of Rho GDP-dissociation inhibitor directs N-terminal inhibitory peptide to GTPases
20. Structural requirements for PAK activation by Rac GTPases