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Volumn 428, Issue 2, 2001, Pages 251-259
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Intracellular alkalinization augments α1-adrenoceptor-mediated vasoconstriction by promotion of Ca2+ entry through the non-L-type Ca2+ channels
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Author keywords
Adrenoceptor agonist; Ammonium chloride; Ca2+ channel; Cytosolic alkalosis; Na+ H+ exchange; Receptor operated; Smooth muscle; Vascular
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Indexed keywords
ALPHA 1 ADRENERGIC RECEPTOR;
ALPHA ADRENERGIC RECEPTOR STIMULATING AGENT;
AMMONIUM CHLORIDE;
CALCIUM CHANNEL;
CALCIUM ION;
CLONIDINE;
NORADRENALIN;
PHENYLEPHRINE;
POTASSIUM CHLORIDE;
PROPIONIC ACID;
VERAPAMIL;
ALKALINIZATION;
ANIMAL EXPERIMENT;
ANIMAL TISSUE;
AORTA;
AORTA STRIP;
ARTERY CONSTRICTION;
ARTICLE;
CALCIUM CELL LEVEL;
CALCIUM TRANSPORT;
CONTROLLED STUDY;
GUINEA PIG;
INCUBATION TIME;
MALE;
MUSCLE CONTRACTILITY;
NONHUMAN;
PH;
PRIORITY JOURNAL;
SMOOTH MUSCLE FIBER;
ADRENERGIC ALPHA-AGONISTS;
AMMONIUM CHLORIDE;
ANIMALS;
AORTA;
CALCIUM;
CALCIUM CHANNEL BLOCKERS;
CALCIUM CHANNELS, L-TYPE;
CLONIDINE;
DOSE-RESPONSE RELATIONSHIP, DRUG;
DRUG SYNERGISM;
GUINEA PIGS;
HYDROGEN-ION CONCENTRATION;
MALE;
MUSCLE CONTRACTION;
MUSCLE, SMOOTH, VASCULAR;
NOREPINEPHRINE;
PHENYLEPHRINE;
PHORBOL 12,13-DIBUTYRATE;
POTASSIUM CHLORIDE;
PROPIONATES;
RECEPTORS, ADRENERGIC, ALPHA-1;
VASOCONSTRICTION;
VERAPAMIL;
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EID: 0035812622
PISSN: 00142999
EISSN: None
Source Type: Journal
DOI: 10.1016/S0014-2999(01)01293-6 Document Type: Article |
Times cited : (7)
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References (32)
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