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Volumn 20, Issue 15, 2001, Pages 1852-1859
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Activation of the Fas pathway independently of Fas ligand during apoptosis induced by camptothecin in p53 mutant human colon carcinoma cells
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Author keywords
Bax; Caspase; FADD; Nuclease; Programmed cell death; z VAD
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Indexed keywords
BENZYLOXYCARBONYLVALYLALANYLASPARTYL FLUOROMETHYL KETONE;
CAMPTOTHECIN;
CASPASE 3;
CYCLOHEXIMIDE;
DNA FRAGMENT;
DNA TOPOISOMERASE;
FAS ANTIBODY;
FAS ANTIGEN;
FAS LIGAND;
NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE;
PROTEIN BAX;
PROTEIN P21;
PROTEIN P53;
APOPTOSIS;
ARTICLE;
CARCINOMA CELL;
CELL MUTANT;
CELL PROTECTION;
COLON CARCINOMA;
CONTROLLED STUDY;
DNA DAMAGE;
GENE ACTIVATION;
GENE EXPRESSION;
HUMAN;
HUMAN CELL;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
ADAPTOR PROTEINS, SIGNAL TRANSDUCING;
AMINO ACID CHLOROMETHYL KETONES;
ANTIGENS, CD95;
ANTINEOPLASTIC AGENTS, PHYTOGENIC;
APOPTOSIS;
CAMPTOTHECIN;
CARRIER PROTEINS;
COLONIC NEOPLASMS;
CYCLOHEXIMIDE;
DNA FRAGMENTATION;
FAS LIGAND PROTEIN;
FAS-ASSOCIATED DEATH DOMAIN PROTEIN;
GENES, P53;
HT29 CELLS;
HUMANS;
MEMBRANE GLYCOPROTEINS;
MUTATION;
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EID: 0035810046
PISSN: 09509232
EISSN: None
Source Type: Journal
DOI: 10.1038/sj.onc.1204264 Document Type: Article |
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Times cited : (86)
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References (44)
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