Reduction of voltage-operated sodium currents by the anticonvulsant drug sulthiame

Brain Research

Volumn 900, Issue 1, 2001, Pages 88-94

  Madeja, Michael ^a,b   Wolf, Christian ^c   Speckmann, Erwin Josef ^a  

^a UNIVERSITY OF MÜNSTER   (Germany)

^b Gemeinnutzige Hertie Stiftung   (Germany)

^c ALK SCHERAX Arzneimittel GmbH   (Germany)

Author keywords

Anticonvulsant drug; Carbonic anhydrase; Disorders of the nervous system; Epilepsy: anticonvulsant drugs; Isolated neuron; Sodium channel; Voltage gated ion channel

Indexed keywords

ANTICONVULSIVE AGENT; CARBONATE DEHYDRATASE; SODIUM CHANNEL; SULTIAME; TETRODOTOXIN;

ACTION POTENTIAL; ANIMAL CELL; ARTICLE; CHANNEL GATING; CONCENTRATION RESPONSE; CONTROLLED STUDY; DRUG EFFECT; GUINEA PIG; NERVE CELL; NONHUMAN; PATCH CLAMP; POTASSIUM CURRENT; PRIORITY JOURNAL; SODIUM CURRENT;

ACTION POTENTIALS; ANIMALS; ANTICONVULSANTS; GUINEA PIGS; HIPPOCAMPUS; ION TRANSPORT; NEURONS; PATCH-CLAMP TECHNIQUES; POTASSIUM; POTASSIUM CHANNEL BLOCKERS; PYRAMIDAL CELLS; SODIUM; SODIUM CHANNEL BLOCKERS; THIAZINES;

EID: 0035805094     PISSN: 00068993     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0006-8993(01)02262-4     Document Type: Article

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