Requirement of the poly(A) tail in coronavirus genome replication

Advances in Experimental Medicine and Biology

Volumn 494, Issue , 2001, Pages 467-474

  Spagnolo, Jeannie F ^a   Hogue, Brenda G ^a  

^a BAYLOR COLLEGE OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

3' UNTRANSLATED REGION; 5' UNTRANSLATED REGION; CONFERENCE PAPER; CORONAVIRUS; MOLECULAR INTERACTION; NONHUMAN; PRIORITY JOURNAL; RNA ANALYSIS; SEQUENCE ANALYSIS; VIRUS GENOME; VIRUS REPLICATION;

3' UNTRANSLATED REGIONS; ANIMALS; CATTLE; CELL LINE; CORONAVIRUS, BOVINE; DEFECTIVE VIRUSES; GENOME, VIRAL; MICE; MURINE HEPATITIS VIRUS; POLY(A)-BINDING PROTEINS; RNA, MESSENGER; RNA-BINDING PROTEINS; VIRUS REPLICATION;

CORONAVIRUS;

EID: 0035703841     PISSN: 00652598     EISSN: None     Source Type: Journal    
DOI: 10.1007/978-1-4615-1325-4_68     Document Type: Article

References (14)

1. cis Requirement for N-specific protein sequence in bovine coronavirus defective interfering RNA replication
2. A cis-acting function for the coronavirus leader in defective interfering RNA replication
3. Binding of encephalomyocarditis virus RNA polymerase to the 3′-noncoding region of the viral RNA is specific and requires the 3′-poly(A) tail
4. The fitness of defective interfering murine coronavirus DI-a and its derivatives is decreased by nonsense and frameshift mutations
5. RNA-RNA recombination in Sindbis virus: Roles of the 3′ conserved motif, poly(A) tail, and nonviral sequences of template RNAs in polymerase recognition and template switching
6. The 5′ end of coronavirus minus-strand RNAs contains a short poly(U) tract
7. Analysis of cis-acting sequences essential for coronavirus defective interfering RNA replication
8. Mouse hepatitis virus A59: Mrna structure and genetic localization of the sequence divergence from hepatotropic strain MHV-3
9. Deletion mapping of a mouse hepatitis virus defective interfering RNA reveals the requirement of an internal and discontiguous sequence for replication
10. Identification of the cis-acting signal for minus-strand RNA synthesis of a murine coronavirus: Implications for the role of minus-strand RNA in RNA replication and transcription
11. Role of 3′-end sequences in infectivity of poliovirus transcripts made in vitro
12. Host protein interactions with the 3′ end of bovine coronavirus RNA and the requirement of the Poly(A) tail for coronavirus defective genome replication
13. Requirement of 3′-terminal poly(adenylic acid) for the infectivity of poliovirus RNA
14. Translation but not the encoded sequence is essential for the efficient propagation of the defective interfering RNAs of the coronavirus mouse hepatitis virus