Mouse hepatitis virus minus-strand templates are unstable and turnover during viral replication

Advances in Experimental Medicine and Biology

Volumn 494, Issue , 2001, Pages 491-497

  Wang, Tao ^a   Sawicki, Stanley G ^a  

^a MEDICAL COLLEGE OF OHIO   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

VIRUS RNA;

ANIMAL CELL; CONFERENCE PAPER; CONTROLLED STUDY; DNA TEMPLATE; GENETIC TRANSCRIPTION; GENOME; HEPATITIS; HEPATITIS VIRUS; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN SYNTHESIS; RNA SYNTHESIS; RNA TRANSLATION; VIRULENCE; VIRUS REPLICATION; VIRUS TRANSCRIPTION;

ANIMALIA; MURINE HEPATITIS VIRUS; RNA VIRUSES;

EID: 0035701557     PISSN: 00652598     EISSN: None     Source Type: Book Series    
DOI: 10.1007/978-1-4615-1325-4_71     Document Type: Conference Paper

References (9)

1. The replication of picornaviruses
2. Subgenomic minus-strand RNA function during mouse hepatitis virus infection
3. Replication of semliki forest virus: Polyadenylate in plus strand RNA and polyuridylate in minus strand RNA
4. Coronavirus minus-strand RNA synthesis and effect of cycloheximide on coronavirus RNA synthesis
5. Coronavirus transcription: Subgenomic mouse hepatitis virus replicative intermediates function in RNA synthesis
6. Coronaviruses use discontinuous extension for synthesis of subgenome-length negative strands
7. A new model for coronavirus transcription
8. Genetics of mouse hepatitis virus transcription: Evidence that subgenomic minus strands are functional templates