Molecular changes in thyroid neoplasia

Folia Histochemica et Cytobiologica

Volumn 39, Issue SUPPL. 2, 2001, Pages 26-27

  Jarzab, B ^a   Woch, J ^a   Wiench, M ^a  

^a INSTITUTE OF ONCOLOGY   (Poland)

Author keywords

Follicular thyroid carcinoma; Papillary thyroid carcinoma; RAS protooncogene; RET protooncogene

Indexed keywords

DNA; PROTEIN P16; PROTEIN P53; PROTEIN RET; RAS PROTEIN;

ANEUPLOIDY; CANCER CLASSIFICATION; CARCINOGENESIS; CHERNOBYL ACCIDENT; CHILD CARE; CLINICAL FEATURE; COMPARATIVE STUDY; CONFERENCE PAPER; DNA METHYLATION; GENE ACTIVATION; GENE FREQUENCY; GENE LOSS; GENE MUTATION; GENE REARRANGEMENT; GENETIC ANALYSIS; GENETIC REGULATION; HETEROZYGOSITY LOSS; HISTOPATHOLOGY; HUMAN; IRRADIATION; MICROSATELLITE INSTABILITY; MOLECULAR BIOLOGY; MORBIDITY; ONCOGENE RAS; ONCOGENE RET; PHENOTYPE; POLAND; SIGNAL TRANSDUCTION; THYROID CANCER; THYROID FOLLICULAR CARCINOMA; THYROID PAPILLARY CARCINOMA; THYROID TUMOR; TUMOR GROWTH; TUMOR SUPPRESSOR GENE; UNDIFFERENTIATED CARCINOMA;

EID: 0035686117     PISSN: 02398508     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Conference Paper

References (15)

1. Pathogenesis of thyroid carcinoma
2. Expression of thyrotropin receptor gene in thyroid carcinoma is associated with good prognosis
3. Mutational activation of ras and gsp oncogenes if differentiated thyroid cancer and their biological implications
4. Frequent loss of heterozygosity on chromosomes 3p and 17p without VHL or p53 mutations suggests involvement of unidentified tumor suppressor genes in follicular thyroid carcinoma
5. High prevalence of mutations of the p53 gene in poorly differentiated human thyroid carcinomas
6. Genetics of follicular thyroid cancer
7. Molecular pathogenesis of tumors of thyroid follicular cells
8. Somatic mutations in the thyrotropin receptor gene cause hyperfunctioning thyroid adenomas
9. High frequency of ras oncogene activation in all stages of human thyroid tumorigenesis
10. Molecular genetics of childhood papillary thyroid carcinomas after irradiation: High prevalence of RET rearrangements
11. Point mutations of ras oncogenes are an early event in thyroid tumorigenesis
12. Involvement of RET oncogene in human tumours: Specificity of RET activation to thyroid tumours
13. Papillary thyroid carcinoma as age-related lesion
14. The RET proto-oncogene in human cancers
15. Age-related activation of the tyrosine-kinase receptor protooncogenes RET and NTRK1 in papillary thyroid carcinoma