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Volumn 20, Issue 50, 2001, Pages 7425-7429
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The defective transforming phenotype of c-Jun Ala63/73 is rescued by mutation of the C-terminal phosphorylation site
a a b a a a c b a |
Author keywords
c Jun transcription factor phosphorylation sites; CBP interaction; Oncogene cooperation; Rat embryo fibroblasts
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Indexed keywords
CYCLIC AMP RESPONSIVE ELEMENT BINDING PROTEIN;
MUTANT PROTEIN;
PROTEIN C JUN;
RAS PROTEIN;
TRANSCRIPTION FACTOR;
ANIMAL CELL;
ARTICLE;
CARBOXY TERMINAL SEQUENCE;
CARCINOGENESIS;
EMBRYO;
ENZYME ACTIVITY;
GENE ACTIVATION;
GENE MUTATION;
GENETIC TRANSCRIPTION;
GENETIC TRANSFECTION;
GENETIC TRANSFORMATION;
NONHUMAN;
ONCOGENE;
PRIORITY JOURNAL;
PROTEIN BINDING;
PROTEIN INTERACTION;
PROTEIN PHOSPHORYLATION;
PROTEIN PROTEIN INTERACTION;
RAT;
TRANSACTIVATION;
ALANINE;
ANIMALS;
CELL LINE, TRANSFORMED;
CELL TRANSFORMATION, NEOPLASTIC;
CYCLIC AMP RESPONSE ELEMENT-BINDING PROTEIN;
DNA;
FIBROBLASTS;
GENES, JUN;
GENES, RAS;
GENES, REPORTER;
GENETIC COMPLEMENTATION TEST;
MICE;
MUTAGENESIS, SITE-DIRECTED;
PHOSPHORYLATION;
PROTEIN PROCESSING, POST-TRANSLATIONAL;
PROTO-ONCOGENE PROTEINS C-JUN;
PROTO-ONCOGENE PROTEINS P21(RAS);
RATS;
RECOMBINANT FUSION PROTEINS;
STRUCTURE-ACTIVITY RELATIONSHIP;
TRANSFECTION;
TUMOR STEM CELL ASSAY;
ANIMALIA;
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EID: 0035504602
PISSN: 09509232
EISSN: None
Source Type: Journal
DOI: 10.1038/sj.onc.1204924 Document Type: Article |
Times cited : (6)
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References (17)
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