Impaired wound healing in transgenic mice overexpressing the activin antagonist follistatin in the epidermis

EMBO Journal

Volumn 20, Issue 19, 2001, Pages 5361-5372

  Wankell, Miriam ^a,b   Munz, Barbara ^b,c   Hübner, Griseldis ^b,d   Hans, Wolfgang ^e   Wolf, Eckhard ^f   Goppelt, Andreas ^e   Werner, Sabine ^a,b  

^a ETH ZURICH   (Switzerland)

^b MAX PLANCK INSTITUTE OF BIOCHEMISTRY   (Germany)

^c STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

^d IQVIA   (United States)

^e SWITCH Biotech AG   (Germany)

^f UNIVERSITY OF MUNICH   (Germany)

Author keywords

Activin; Epidermis; Follistatin; Keratinocyte; Wound

Indexed keywords

ACTIVIN; FOLLISTATIN;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; DERMIS; EPIDERMIS; GRANULATION TISSUE; MOUSE; NONHUMAN; NUCLEOTIDE SEQUENCE; PATHOGENESIS; PRIORITY JOURNAL; PROTEIN EXPRESSION; SCAR; SCAR FORMATION; SKIN ATROPHY; TRANSGENIC MOUSE; WOUND DEHISCENCE; WOUND HEALING; WOUND HEALING IMPAIRMENT;

ACTIVINS; ANIMALS; BONE MORPHOGENETIC PROTEINS; CELL DIFFERENTIATION; CELL DIVISION; CLONING, MOLECULAR; EAR; EPIDERMIS; FOLLISTATIN; GENE EXPRESSION REGULATION; GROWTH SUBSTANCES; KERATINOCYTES; MICE; MICE, TRANSGENIC; SKIN; SKIN ABNORMALITIES; SKIN PHYSIOLOGY; TRANSGENES; WOUND HEALING;

EID: 0035477312     PISSN: 02614189     EISSN: None     Source Type: Journal    
DOI: 10.1093/emboj/20.19.5361     Document Type: Article

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