Kainic acid induces selective mitochondrial oxidative phosphorylation enzyme dysfunction in cerebellar granule neurons: protective effects of melatonin and GSH ethyl ester.

The FASEB journal : official publication of the Federation of American Societies for Experimental Biology

Volumn 15, Issue 10, 2001, Pages 1786-1788

  Dabbeni Sala, F ^a   Floreani, M ^a   Franceschini, D ^a   Skaper, S D ^a   Giusti, P ^a  

^a UNIVERSITY OF PADOVA   (Italy)

Author keywords

[No Author keywords available]

Indexed keywords

ANTIOXIDANT; DRUG DERIVATIVE; GLUTATHIONE; KAINIC ACID; MELATONIN; MULTIENZYME COMPLEX; OXIDOREDUCTASE; REACTIVE OXYGEN METABOLITE; S ETHYL GLUTATHIONE; S-ETHYL GLUTATHIONE; SUCCINATE DEHYDROGENASE; SUCCINATE DEHYDROGENASE (UBIQUINONE);

ANIMAL; ARTICLE; CELL SURVIVAL; CEREBELLUM; CYTOLOGY; DRUG EFFECT; ENZYMOLOGY; METABOLISM; MITOCHONDRION; NERVE CELL; OXIDATIVE PHOSPHORYLATION; RAT; ULTRASTRUCTURE;

ANIMALS; ANTIOXIDANTS; CELL SURVIVAL; CEREBELLUM; ELECTRON TRANSPORT COMPLEX II; GLUTATHIONE; KAINIC ACID; MELATONIN; MITOCHONDRIA; MULTIENZYME COMPLEXES; NEURONS; OXIDATIVE PHOSPHORYLATION; OXIDOREDUCTASES; RATS; REACTIVE OXYGEN SPECIES; SUCCINATE DEHYDROGENASE;

MLCS; MLOWN;

EID: 0035432155     PISSN: 08926638     EISSN: None     Source Type: Journal    
DOI: 10.1096/fj.00-0427fje     Document Type: Article

References (0)