A mathematical model for the dynamics of HIV-1 during the typical course of infection

Nonlinear Analysis, Theory, Methods and Applications

Volumn 47, Issue 3, 2001, Pages 1773-1783

  Gumel, A B ^a,b   Shivakumar, P N ^a,b   Sahai, B M ^a,b  

^a UNIVERSITY OF MANITOBA   (Canada)

^b NONE

Author keywords

CD4+ T cells; Finite difference; Replication; Stability

Indexed keywords

BLOOD; CYTOLOGY; FINITE DIFFERENCE METHOD; LINEARIZATION; MATHEMATICAL MODELS; PATHOLOGY; VIRUSES;

DISEASE PROGRESSION;

IMMUNOLOGY;

EID: 0035425119     PISSN: 0362546X     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0362-546X(01)00309-1     Document Type: Conference Paper

References (10)

1. A nuclear localization signal within HIV-1 matrix protein that governs infection of non-dividing cells
2. GM-CSF and its effects on replication of HIV-1 in cells of macrophage lineage
3. Changes in thymic function with age and during the treatment of HIV infection
4. + T-cell subpopulations
5. A model for treatment strategy in the chemotherapy of AIDS
6. AIDS: Background and the dynamics of the decline of immunocompetence
7. Mathematical analysis of HIV-1 dynamics in vivo
8. HIV-1 replication is controlled at the level of T cell activation and proviral integration
9. Numerical methods, with applications in the biomedical sciences
10. Incompletely reverse-transcribed human immunodeficiency virus type 1 genomes in quiescent cells can function as intermediates in retroviral cycle