How can anti-diabetics suppress tumours?

Trends in Pharmacological Sciences

Volumn 22, Issue 8, 2001, Pages 399-

  Wood, Ian ^a  

^a NONE

Author keywords

[No Author keywords available]

Indexed keywords

ANTIDIABETIC AGENT; PEROXISOME PROLIFERATOR; PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR GAMMA; ROSIGLITAZONE;

APOPTOSIS; CANCER CELL; CANCER CHEMOTHERAPY; CANCER INHIBITION; CELL DIFFERENTIATION; CELL PROLIFERATION; CHROMOSOME 10; CONCENTRATION RESPONSE; DNA RESPONSIVE ELEMENT; GENE ACTIVITY; GENE EXPRESSION REGULATION; GENE LOSS; GENE SEQUENCE; GENETIC TRANSCRIPTION; HUMAN; MACROPHAGE; MONOCYTE; NOTE; PRIORITY JOURNAL; PROMOTER REGION; RECEPTOR AFFINITY; SEQUENCE ANALYSIS; TUMOR SUPPRESSOR GENE;

EID: 0035423596     PISSN: 01656147     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0165-6147(00)01790-9     Document Type: Short Survey

References (1)

1. Tumor suppressor and anti-inflammatory actions of PPARγ agonists are mediated via upregulation of PTEN