Interaction of disintegrins with human neutrophils induces cytoskeleton reorganization, focal adhesion kinase activation, and extracellular-regulated kinase-2 nuclear translocation, interfering with the chemotactic function.

The FASEB journal : official publication of the Federation of American Societies for Experimental Biology

Volumn 15, Issue 9, 2001, Pages 1643-1645

  Coelho, A L ^a   De Freitas, M S ^a   Mariano Oliveira, A ^a   Oliveira Carvalho, A L ^a   Zingali, R B ^a   Barja Fidalgo, C ^a  

^a STATE UNIVERSITY OF RIO DE JANEIRO   (Brazil)

Author keywords

[No Author keywords available]

Indexed keywords

ACTIN; DISINTEGRIN; FOCAL ADHESION KINASE; FOCAL ADHESION KINASE 1; MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE 1; PROTEIN TYROSINE KINASE; PTK2 PROTEIN, HUMAN;

ACTIVE TRANSPORT; ARTICLE; CHEMOTAXIS; CYTOSKELETON; DRUG EFFECT; ENZYME ACTIVATION; ENZYMOLOGY; HUMAN; IN VITRO STUDY; METABOLISM; NEUTROPHIL; PHYSIOLOGY; SIGNAL TRANSDUCTION;

ACTINS; ACTIVE TRANSPORT, CELL NUCLEUS; CHEMOTAXIS; CYTOSKELETON; DISINTEGRINS; ENZYME ACTIVATION; FOCAL ADHESION KINASE 1; FOCAL ADHESION PROTEIN-TYROSINE KINASES; HUMANS; MITOGEN-ACTIVATED PROTEIN KINASE 1; MITOGEN-ACTIVATED PROTEIN KINASES; NEUTROPHILS; PROTEIN-TYROSINE KINASES; SIGNAL TRANSDUCTION;

MLCS; MLOWN;

EID: 0035408295     PISSN: 08926638     EISSN: None     Source Type: Journal    
DOI: 10.1096/fj.00-0812fje     Document Type: Article

References (0)