Dystrophin-deficient cardiomyocytes are abnormally vulnerable to mechanical stress-induced contractile failure and injury.

The FASEB journal : official publication of the Federation of American Societies for Experimental Biology

Volumn 15, Issue 9, 2001, Pages 1655-1657

  Danialou, G ^a   Comtois, A S ^a   Dudley, R ^a   Karpati, G ^a   Vincent, G ^a   Des Rosiers, C ^a   Petrof, B J ^a  

^a MCGILL UNIVERSITY HEALTH CENTRE   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

DYSTROPHIN;

ANIMAL; ARTICLE; C57BL MOUSE; HEART; HEART CONTRACTION; HEART LEFT VENTRICLE FUNCTION; HEART MUSCLE; IN VITRO STUDY; MECHANICAL STRESS; METABOLISM; MORTALITY; MOUSE; PATHOLOGY; PATHOPHYSIOLOGY; PERFUSION; PHYSIOLOGY;

ANIMALS; DYSTROPHIN; HEART; MICE; MICE, INBRED C57BL; MYOCARDIAL CONTRACTION; MYOCARDIUM; PERFUSION; STRESS, MECHANICAL; VENTRICULAR DYSFUNCTION, LEFT;

MLCS; MLOWN;

EID: 0035403114     PISSN: 08926638     EISSN: None     Source Type: Journal    
DOI: 10.1096/fj.01-0030fje     Document Type: Article

References (0)