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Volumn 7, Issue 3, 2001, Pages 235-241
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Bax gene disruption alters the epidermal response to ultraviolet irradiation and in vivo induced skin carcinogenesis.
a
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Author keywords
[No Author keywords available]
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Indexed keywords
7,12 DIMETHYLBENZ[A]ANTHRACENE;
BAX PROTEIN, MOUSE;
CARCINOGEN;
KERATIN;
LORICRIN;
MEMBRANE PROTEIN;
ONCOPROTEIN;
PHORBOL 13 ACETATE 12 MYRISTATE;
PROTEIN BAX;
PROTEIN BCL 2;
ANIMAL;
APOPTOSIS;
ARTICLE;
CELL DIFFERENTIATION;
CHEMICALLY INDUCED DISORDER;
CYTOLOGY;
EPIDERMIS;
GENETICS;
IMMUNOBLOTTING;
IMMUNOHISTOCHEMISTRY;
KERATINOCYTE;
METABOLISM;
MOUSE;
MOUSE MUTANT;
NICK END LABELING;
PATHOPHYSIOLOGY;
PHYSIOLOGY;
PROTO ONCOGENE;
RADIATION EXPOSURE;
SKIN TUMOR;
ULTRAVIOLET RADIATION;
9,10-DIMETHYL-1,2-BENZANTHRACENE;
ANIMALS;
APOPTOSIS;
BCL-2-ASSOCIATED X PROTEIN;
CARCINOGENS;
CELL DIFFERENTIATION;
EPIDERMIS;
GENES, BCL-2;
IMMUNOBLOTTING;
IMMUNOHISTOCHEMISTRY;
IN SITU NICK-END LABELING;
KERATINOCYTES;
KERATINS;
MEMBRANE PROTEINS;
MICE;
MICE, KNOCKOUT;
PROTO-ONCOGENE PROTEINS;
PROTO-ONCOGENE PROTEINS C-BCL-2;
SKIN NEOPLASMS;
TETRADECANOYLPHORBOL ACETATE;
ULTRAVIOLET RAYS;
MLCS;
MLOWN;
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EID: 0035286182
PISSN: 11073756
EISSN: None
Source Type: Journal
DOI: 10.3892/ijmm.7.3.235 Document Type: Article |
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Times cited : (24)
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References (0)
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