Human pancreatic cancer cells disable function of Fas receptors at several levels in Fas signal transduction pathway.

International journal of oncology

Volumn 18, Issue 2, 2001, Pages 311-316

  Elnemr, A ^a   Ohta, T ^a   Yachie, A ^a   Kayahara, M ^a   Kitagawa, H ^a   Fujimura, T ^a   Ninomiya, I ^a   Fushida, S ^a   Nishimura, G I ^a   Shimizu, K ^a   Miwa, K ^a  

^a KANAZAWA UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

CARRIER PROTEIN; CASP8 PROTEIN, HUMAN; CASP9 PROTEIN, HUMAN; CASPASE; CASPASE 8; CASPASE 9; CFLAR PROTEIN, HUMAN; FAS ANTIGEN; FLICE INHIBITORY PROTEIN; MESSENGER RNA; PROTEIN TYROSINE PHOSPHATASE; PTPN13 PROTEIN, HUMAN; SIGNAL PEPTIDE; TUMOR PROTEIN;

ADENOCARCINOMA; APOPTOSIS; ARTICLE; CELL CULTURE; HUMAN; METABOLISM; PANCREAS TUMOR; PHYSIOLOGY; SIGNAL TRANSDUCTION;

ADENOCARCINOMA; ANTIGENS, CD95; APOPTOSIS; CARRIER PROTEINS; CASP8 AND FADD-LIKE APOPTOSIS REGULATING PROTEIN; CASPASE 8; CASPASE 9; CASPASES; HUMANS; INTRACELLULAR SIGNALING PEPTIDES AND PROTEINS; NEOPLASM PROTEINS; PANCREATIC NEOPLASMS; PROTEIN-TYROSINE-PHOSPHATASE; RNA, MESSENGER; SIGNAL TRANSDUCTION; TUMOR CELLS, CULTURED;

MLCS; MLOWN;

EID: 0035258678     PISSN: 10196439     EISSN: None     Source Type: Journal    
DOI: 10.3892/ijo.18.2.311     Document Type: Article

References (0)