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Volumn 17, Issue 2, 2001, Pages 83-88

Too much of a good thing: Mechanisms of gene action in Down syndrome

Author keywords

[No Author keywords available]

Indexed keywords

ANEUPLOIDY; ANIMAL MODEL; DOWN SYNDROME; GENE EXPRESSION REGULATION; GENE FUNCTION; HUMAN; MOUSE; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; REVIEW; TRANSGENIC MOUSE; TRISOMY 21;

EID: 0035252562     PISSN: 01689525     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0168-9525(00)02172-7     Document Type: Review
Times cited : (127)

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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.