Apoptosis during spermatogenesis: The thrill of being alive

Molecular Reproduction and Development

Volumn 58, Issue 1, 2001, Pages 1-3

  Kierszenbaum, Abraham L ^a,b  

^a CITY UNIVERSITY OF NEW YORK MEDICAL SCHOOL   (United States)

^b CITY UNIVERSITY OF NEW YORK   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE;

APOPTOSIS; ARTICLE; CELL DIFFERENTIATION; ENZYME ACTIVATION; IMMUNE SYSTEM; NONHUMAN; PRIORITY JOURNAL; SEMINIFEROUS TUBULE; SERTOLI CELL; SIGNAL TRANSDUCTION; SPERMATOCYTE; SPERMATOGENESIS; TESTIS;

ANIMALS; ANTIGENS, CD95; APOPTOSIS; CASPASES; CELL SURVIVAL; FAS LIGAND PROTEIN; MEMBRANE GLYCOPROTEINS; PROTO-ONCOGENE PROTEINS C-BCL-2; SPERMATOGENESIS;

EID: 0035205090     PISSN: 1040452X     EISSN: None     Source Type: Journal    
DOI: 10.1002/1098-2795(200101)58:1<1::AID-MRD1>3.0.CO;2-0     Document Type: Article

References (7)

1. Targeted mutation in the Fas gene causes hyperplasia in peripheral lymphoid organs and liver
2. Fas and Fas ligand in embryos and adult mice: Ligand expression in several immune-privileged tissues and coexpression in adult tissues characterized by apoptotic cell turnover
3. Inhibition of testicular germ cell apoptosis and differentiation in mice misexpressing Bcl-2 in spermatogenesis
4. BCL2 protein is topographically restricted in tissues characterized by apoptotic cell death
5. Apoptosis regulator Bcl-w is essential for spermatogenesis but appears otherwise redundant
6. An early and massive wave of germinal cell apoptosis is required for the development of functional spermatogenesis
7. Cell death patterns of the rat spermatogonial cell progeny induced by Sertoli cell geometric changes and Fas (CD95) agonist